Otological and vestibular symptoms in patients with low grade (Quebec grades one and two) whiplash injury

Rowlands, R.G.; Campbell, Islay; Kenyon, Guy

doi:10.1017/s0022215108002569

Cited by 17 publications

(8 citation statements)

References 18 publications

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“…95 There is a remarkable difference between the reported prevalence (range, 10%-15%) of otological symptoms several months after whiplash injury. 97 These data point to a delay in the development of tinnitus after neck trauma. 97 These data point to a delay in the development of tinnitus after neck trauma.…”

Section: Whiplash/neck Traumamentioning

confidence: 97%

Trauma-Associated Tinnitus

Kreuzer¹,

Landgrebe²,

Vielsmeier³

et al. 2014

Journal of Head Trauma Rehabilitation

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Section: Whiplash/neck Traumamentioning

confidence: 97%

Trauma-Associated Tinnitus

Kreuzer¹,

Landgrebe²,

Vielsmeier³

et al. 2014

Journal of Head Trauma Rehabilitation

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“…63 One study of individuals evaluated for legal claims from whiplash injury reported a very low proportion (3.7%) having any vestibular symptoms or complaints. 64 It may be that the actual physical trauma of a mTBI is a less important factor than is female gender and the presence of preinjury depression and anxiety, posttraumatic stress disorder, and pain, which predispose to prolonged postconcussive syndrome symptoms. [65][66][67] As one hypothesis goes, mild head trauma may be the initiating factor, but it is the pre-existing psychological condition that causes the prolonged postconcussive syndrome symptoms.…”

Section: Prognosismentioning

confidence: 99%

Posttraumatic Vertigo and Dizziness

Fife

Giza

2013

Semin Neurol

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Dizziness and vertigo are common symptoms following minor head trauma. Although these symptoms resolve within a few weeks in many patients, in some the symptoms may last much longer and impede ability to return to work and full functioning. Causes of persisting or recurrent dizziness may include benign paroxysmal positional vertigo, so-called labyrinthine concussion, unilateral vestibular nerve injury or damage to the utricle or saccule, perilymphatic fistula, or less commonly traumatic endolymphatic hydrops. Some dizziness after head trauma is due to nonlabyrinthine causes that may be related to structural or microstructural central nervous system injury or to more complicated interactions between migraine, generalized anxiety, and issues related to patients self-perception, predisposing psychological states, and environmental and stress-related factors. In this article, the authors review both the inner ear causes of dizziness after concussion and also the current understanding of chronic postconcussive dizziness when no peripheral vestibular cause can be identified.

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“…Vehicle collisions rear-end shunts have lead to whiplash injuries to the cervical spine that have been associated with unilateral tinnitus developing immediately following the injury (Rowlands et al, 2009).…”

Section: Bannister Fairfi Eld General Hospital Bury Ukmentioning

confidence: 99%

Abstracts of the British Society of Audiology annual conference (incorporating the Experimental and Clinical Short papers meetings)

2013

International Journal of Audiology

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Tinnitus, the sensation of sound in the absence of an external sound source, is primarily associated with noise exposure and ageing. Tinnitus is a huge burden to the population ( Ͼ 5 million in the UK), but it is a complex condition to manage clinically. The absence of an effective pharmacological cure leaves the patient with the prospect of adjusting to this abnormal sensation, as well as to any psychological co-morbidity. Effective treatments need to address multiple component factors including feelings of intrusiveness, reduced sense of control, cognitive interference, sleep disturbance, auditory difficulties attributed to tinnitus, interference with relaxation, emotional distress and reduced quality of life.The fi eld has gained a rather unfortunate reputation for poor quality research, some examples of which were eloquently presented in Prof Brian Moore ' s Twilight lecture given at the fi rst combined BSA conference in 2010. However, tinnitus research is currently enjoying somewhat of a renaissance with a new generation of scientists and clinicians working actively on questions relating to basic mechanisms, treatment effi cacy and health service provision. Since our Nottingham translational research programme in tinnitus was established in 2008/9, our research strategy has begun to address several key themes (as discussed by Chalmers and Glasziou, 2009):(1) Helping to ensure that those who fund health research are aware of what matters to both patients and clinicians. Public funding of research is correlated only modestly with disease burden, if at all. Within specifi c health problems there is little research on the extent to which questions addressed by researchers match questions of relevance to patients and clinicians. A current initiative, supported by the James Lind Alliance and NIHR, brings together academics, clinicians and patients to identify and prioritise uncertainties relating to cause, assessment and treatment of tinnitus.(2) Using appropriate design and methods. It would seem logical that there is a need for new research only if the questions it proposes to address cannot be answered satisfactorily with existing evidence. Yet, few studies are designed with reference to systematic reviews of existing evidence. Where that evidence does not yet exist, we have contributed to the fi eld by conducting those reviews. Our systematic reviews have made specifi c recommendations to guide tinnitus researchers on good practice in trial design including randomised allocation, active control intervention, blinding and post-treatment follow-up.These guiding principles aim to bring a scientifi c rigour and transparency to our tinnitus work in order to further stimulate the recent resurgence of research activity. Our hope is to provide some defi nitive answers to long-standing questions.Current theories of tinnitus assume that the phantom percept is triggered by cochlear damage. However, around 10% of tinnitus patients have a normal audiogram, i.e., no direct signs of cochlear damage, suggesting either that ti...

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Otological and vestibular symptoms in patients with low grade (Quebec grades one and two) whiplash injury

Cited by 17 publications

References 18 publications

Trauma-Associated Tinnitus

Trauma-Associated Tinnitus

Posttraumatic Vertigo and Dizziness

Abstracts of the British Society of Audiology annual conference (incorporating the Experimental and Clinical Short papers meetings)

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