2007
DOI: 10.1016/j.ajem.2006.05.025
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Other causes of unilateral pulmonary edema

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Cited by 15 publications
(9 citation statements)
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“…Leukocytosis has previously been reported in patients with UPE resulting from severe MR in the absence of proven infection. 3,7,8,22 The association of unilateral pulmonary infiltrates and/or leukocytosis and/or acute respiratory distress led to the initiation of antibiotic therapy, usually in the absence of fever. This may delay therapeutic management of UPE; initiation of appropriate treatment was delayed in 33% of our UPE cases versus 4% in patients without UPE (Pϭ0.003), highlighting the difficulty of immediately making the correct diagnosis of UPE.…”
Section: Discussionmentioning
confidence: 99%
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“…Leukocytosis has previously been reported in patients with UPE resulting from severe MR in the absence of proven infection. 3,7,8,22 The association of unilateral pulmonary infiltrates and/or leukocytosis and/or acute respiratory distress led to the initiation of antibiotic therapy, usually in the absence of fever. This may delay therapeutic management of UPE; initiation of appropriate treatment was delayed in 33% of our UPE cases versus 4% in patients without UPE (Pϭ0.003), highlighting the difficulty of immediately making the correct diagnosis of UPE.…”
Section: Discussionmentioning
confidence: 99%
“…The unilateral location of cardiogenic pulmonary edema is unusual [3][4][5][6][7][8] and has rarely been described and mainly reported in association with severe mitral regurgitation (MR). 9,10 Little is known about the characteristics of unilateral pulmonary edema (UPE), and its prevalence has never been assessed in a large series of pulmonary edema.…”
Section: Clinical Perspective On P 1115mentioning
confidence: 99%
“…Unilateral oedema with ipsilateral pathology has been reported with prolonged lateral decubitus position,[6] rapid thoracentesis,[7] after acute upper airway obstruction,[8] pulmonary contusion,[9] talc pleurodesis,[10] pulmonary vein occlusion,[11] and congenital or surgical systemic to pulmonary shunt (e.g., Blalock-Taussig shunt). [12]…”
Section: Discussionmentioning
confidence: 99%
“…It has been reported after congestive heart failure, prolonged rest on one side in patients with cardiac decompensation or receiving large amounts of fluids, in cases of rapid expansion of the lungs after pleural effusion and pneumothorax; it is also seen in the normal lung in patients with unilateral pulmonary disease such as MacLeod syndrome and unilateral pulmonary artery hypoplasia or agenesis, pulmonary artery compression from aortic dissection or left ventricular pseudoaneurysm and pulmonary venous obstruction from mediastinal fibrosis. However, it is mainly reported in association with severe MR (1-4). …”
Section: Discussionmentioning
confidence: 99%