On the mechanism of action of pneumatic compression devices: Combined magnetic resonance imaging and duplex ultrasound investigation

Lurie, Fedor; Scott, Victoria; Yoon, Hyo‐Chun; Kistner, Robert L.

doi:10.1016/j.jvs.2008.04.009

Cited by 32 publications

(32 citation statements)

References 13 publications

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“…Studies have shown that IPC of the lower limbs may improve venous blood flow in the venous system and increase fibrinolytic activity. 51,82 Thus, it is perhaps not surprising to observe that IPC is more effective than no prophylaxis or TEDS in reducing venous thromboembolism. Using IPC alone, however, may not be completely effective in preventing thromboembolism in high-risk patients, combined use of IPC and pharmacological thromboprophylaxis can tackle both venous stasis and hypercoagulability simultaneously.…”

Section: Discussionmentioning

confidence: 99%

Stratified Meta-Analysis of Intermittent Pneumatic Compression of the Lower Limbs to Prevent Venous Thromboembolism in Hospitalized Patients

2013

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Section: Discussionmentioning

confidence: 99%

Stratified Meta-Analysis of Intermittent Pneumatic Compression of the Lower Limbs to Prevent Venous Thromboembolism in Hospitalized Patients

2013

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“…The prevention of stasis is a main goal in CVD treatment and decisive in preventing venous complications and is frequently done through conservative approaches. Conservative CVD treatment might include intermittent pneumatic compression [116], compression stockings and bandages [77,117,118], and muscle pump activation using electrical muscle stimulation [77,119], transcutaneous electrical nerve stimulation [119], or active and passive movements [103,119]. In this regard, MLD maneuvers may be an alternative treatment to enhance venous flow [21][22][23].…”

Section: Conservative Treatment Of Cvdmentioning

confidence: 99%

Manual Lymphatic Drainage in the Treatment of Chronic Venous Disease

Crisóstomo¹,

Armada-da-Silva²

2017

Clinical Physical Therapy

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Chronic venous disease (CVD) is a chronic condition that is associated with venous hypertension, vein's valves damage, venous obstruction, and calf muscle pump impairment. This blood circulatory condition is also characterized by important inflammatory changes affecting the skin, the subcutaneous tissue and the muscles, which are probably triggered by blood stasis and venous edema. With disease progression, severe ulcerative skin damage might occur, which when present represent the more severe stage of this condition. CVD has a significant economic, social and health impact, mostly due to raised morbidity and chronicity.The treatment of patients with CVD might focus on both the symptoms and secondary changes of the disease, such as edema, skin and subcutaneous changes or ulcers.Usually, initial treatment of CVD patients involves a non-invasive, conservative treatment to reduce symptoms, treat secondary changes, and help prevent the development of secondary complications and the progression of the disease. Complementary, some interventional or surgical treatments can be undertaken.There are several conservative treatments to treat and prevent complications associated with CVD that have been described in the literature, like manual lymphatic drainage (MLD) and compression, physical exercise, intermittent pneumatic pressure, kinesio taping, electrical muscle stimulation, transcutaneous electrical nerve stimulation, hydrotherapy, and health education. Most of these techniques are complementary to compression therapy or pharmacological treatment. This chapter will address the role of physical therapists in the management of CVD. The chapter will begin by reviewing the basic physiopathology of CVD, including the role of calf muscle pump. The CEAP classification system and the chronic venous severity score will be presented, as these are main tools for clinical assessment of CVD severity. In the remainder of the chapter will address the physiological effects and recommendations for treating CVD of MLD, based on our clinical experience and own research.

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“…Lurie et al 17 used MRI and Duplex ultrasound to clarify the mode of action of intermittent pneumatic compression, which is more complex to assess than permanent compression. They found a measurable decrease in the volume of subcutaneous tissue under the garment.…”

Section: For the Future: A Numerical Approach And Simulation By Finitmentioning

confidence: 99%

Relationship between medical compression and intramuscular pressure as an explanation of a compression paradox

Cornu‐Thénard

Fournier³

et al. 2014

Phlebology

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We found that an IP of 22 mmHg produced a significant IMP increase from 32 to 54 mmHg, in the standing position. At the same time, the subcutaneous pressure is only provided by the compression device, on healthy subjects. In other words, the subcutaneous pressure plus the IP is only a little higher than 22 mmHg-a pressure which is too low to reduce the caliber of the superficial veins. This is in accordance with our standing MRI 3D anatomical study which showed that, paradoxically, when applying low pressures (IP), the deep veins are compressed while the superficial veins are not.

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On the mechanism of action of pneumatic compression devices: Combined magnetic resonance imaging and duplex ultrasound investigation

Cited by 32 publications

References 13 publications

Stratified Meta-Analysis of Intermittent Pneumatic Compression of the Lower Limbs to Prevent Venous Thromboembolism in Hospitalized Patients

Stratified Meta-Analysis of Intermittent Pneumatic Compression of the Lower Limbs to Prevent Venous Thromboembolism in Hospitalized Patients

Manual Lymphatic Drainage in the Treatment of Chronic Venous Disease

Relationship between medical compression and intramuscular pressure as an explanation of a compression paradox

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