“…3,7,8 A sequence of events occurs in acute and chronic inflammation of many different causes, resulting in the inflammation of the adjacent colonic serosal surface and adhesion to the gallbladder. 1,3,6 The ischemic area in the wall of the ga l lbladder or colon becomes gangrenous, and the increased pressure within results in its contents penetrating its own necrotic wall at first and then the wall of the adjacent organs, forming a CCF. 9,10 A spontaneous CCF comprises 10-20% of all biliary enteric fistulas.…”