Œdème pulmonaire à pression négative après une cholécystectomie – à propos d’un cas

Tebay, A.; Bouti, Khalid; Tebay, N.

doi:10.1016/j.pneumo.2017.08.006

Cited by 7 publications

(8 citation statements)

References 16 publications

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“…Young, healthy, athletic patients seem to be at risk for NPPE. [4] However, one quarter of these reported cases are the patients over 55 years old. Thus, crucial attention should be given to NPPE discovered in old patients.…”

Section: Discussionmentioning

confidence: 99%

“…This occurs in 0.05% to 0.1% of cases as a serious complication of general anesthesia with tracheal intubation, [1,2] and more than half of these cases are related to postanesthetic laryngospasm. [3,4] Postanesthetic NPPE is a potentially life-threatening emergency and occurs more frequently than reported in studies, which should cause the attention for anesthesiologists. Here, we presented a rare case of “delayed” NPPE in a young woman after endoscopic thyroidectomy and reviewed the reported cases of NPPE over the past decade.…”

Section: Introductionmentioning

confidence: 99%

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Negative pressure pulmonary edema after general anesthesia

et al. 2019

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Rationale:Negative pressure pulmonary edema (NPPE) is a dangerous clinical complication and potentially life-threatening emergency without prompt diagnosis and intervention during recovery period after anesthetic extubation.Patient concerns:A 25-year-old woman has undergone endoscopic thyroidectomy. After extubation, the patient developed acute respiratory distress with high airway resistance accompanied with wheezing, oxyhemoglobin saturation (SpO2) decreased to 70%. With positive pressure mask ventilation, her condition was stable, SpO2 99%. However, the patient developed pink frothy sputum with diffuse bilateral rales 30 min later after transported to surgical intensive care unit (SICU).Diagnoses:Negative pressure pulmonary edema.Interventions:The patient was undergone assisted ventilation with continuous positive airway pressure (CPAP) and furosemide 20 mg was given intravenously.Outcomes:Postoperative day (POD) 2 her condition became stable, computed tomography (CT) scan indicated the pulmonary edema disappeared. The patient was discharged 6 days later. No abnormalities were observed during following 4 weeks.Lessons:Although usually the onset of NPPE is rapid, with individual differences NPPE is still challenging. Increased vigilance in monitoring, diagnosis, and treatment are essential to prevent aggravation and further complication.

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Section: Discussionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Negative pressure pulmonary edema after general anesthesia

et al. 2019

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“…negative intrathoracic pressure as high as À140 cm H 2 O, which increases the hydrostatic pressure in the pulmonary capillary bed causing peri-microvascular interstitial and alveolar oedema. 3 NPPE usually develops in children and young adults immediately post-extubation, 4 but can be delayed up to few hours. Non-invasive ventilation and diuretics are usually sufficient to manage NPPE patients.…”

Section: Discussionmentioning

confidence: 99%

Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess

Chaudhry

Nimmala

Papudesi

et al. 2021

Respirology Case Reports

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A 61‐year‐old male presented with progressive generalized weakness, myalgia, diaphoresis, fever, episodic chills and rigors that had started 4 days previously. Chest x‐ray (CXR) showed overlying curvilinear radio‐opacities. Abdominal computed tomography revealed liver and bilateral adrenal lesions. Empiric 7‐day intravenous Piperacillin / Tazobactam (Zosyn) was initiated, and he was admitted for sepsis. After an episode of rigors on Day 2, he developed acute hypoxic respiratory failure with inspiratory stridor. CXR revealed new, bilateral airspace disease. Racemic Epinephrine, Solumedrol, Ketorolac (Toradol) and Diphenhydramine were given, and he was transferred to the intensive care unit with presumptive diagnosis of foreign body aspiration or allergic reaction. With each subsequent episode of rigor and chills, he continued developing hypoxic respiratory failure with stridor and an incremental increase in pulmonary oedema on imaging. Pulmonologist concluded it likely secondary to negative pressure pulmonary oedema caused by transient laryngeal dyskinesia induced by the increased work of breathing associated with rigors. Symptoms resolved after the complete course of antibiotics along with supportive therapy.

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“…10 Type II NPPE usually occurs after relief of chronic airway obstruction, for example, after the operation of tonsillar hypertrophy, adenoid hypertrophy and upper respiratory tract tumours. 12,13 The incidence of NPPE is between 0.1 and 11%, 11,14,15 and it has been reported that the incidence of NPPE in China is as high as 9.6%. 16 The most common cause of NPPE in adults is the laryngospasm after extubation under general anaesthesia, with an incidence of 3% 11,12 ; but the cause of NPPE in children is different from that in adults, which is mainly due to airway obstruction.…”

Section: Discussionmentioning

confidence: 99%

Imaging characteristics and mechanism of negative pressure pulmonary edema in children: three case reports

Liang

2021

Cardiol Young

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This article reports three children who presented with negative pressure pulmonary oedema before or after removal of the airway foreign bodies. Of them, two cases were type I negative pressure pulmonary oedema and one case was type II negative pressure pulmonary oedema following foreign body removal from the airways. Pathogenesis and location differences between type I and type II negative pressure pulmonary oedema caused by airway foreign body obstruction, as well as diagnosis and treatment modalities of the negative pressure pulmonary oedema were discussed.

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Œdème pulmonaire à pression négative après une cholécystectomie – à propos d’un cas

Cited by 7 publications

References 16 publications

Negative pressure pulmonary edema after general anesthesia

Negative pressure pulmonary edema after general anesthesia

Negative pressure pulmonary oedema due to rigors and chills associated with liver abscess

Imaging characteristics and mechanism of negative pressure pulmonary edema in children: three case reports

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