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“…This can be due to changed isoenzyme composition of myosin (the main protein of the thick filament of myofibrils), impaired excitation/contraction coupling, and Ca 2+ homeostasis disorders [5]. Impairment of contractile activity of myocardial myofibrils in hypothyrosis can be explained by low effectivity of the contraction process, disorders in Ca 2+ regulation of the contraction process by the thin filament, and changed tertiary structure of actin [1].…”
mentioning
confidence: 99%
“…This can be due to changed isoenzyme composition of myosin (the main protein of the thick filament of myofibrils), impaired excitation/contraction coupling, and Ca 2+ homeostasis disorders [5]. Impairment of contractile activity of myocardial myofibrils in hypothyrosis can be explained by low effectivity of the contraction process, disorders in Ca 2+ regulation of the contraction process by the thin filament, and changed tertiary structure of actin [1].…”
mentioning
confidence: 99%
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