Not All Acne Is Acne Vulgaris

Gollnick, Harald; Zouboulis, Christos C.

doi:10.3238/arztebl.2014.0301

Cited by 48 publications

(57 citation statements)

References 100 publications

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“…For example, patients being treated with topical therapy/therapies should be moved to systemic therapies. Patients being treated with systemic therapies should change to oral isotretinoin as this will more rapidly reduce inflammation and prevent scarring …”

Section: Resultsmentioning

confidence: 99%

A consensus‐based practical and daily guide for the treatment of acne patients

Gollnick

Bettoli

Lambert

et al. 2016

Acad Dermatol Venereol

106

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Section: Resultsmentioning

confidence: 99%

A consensus‐based practical and daily guide for the treatment of acne patients

Gollnick

Bettoli

Lambert

et al. 2016

Acad Dermatol Venereol

106

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show abstract

“…In acne, multiple phenotypes exist that are not all due to the same pathogenetic processes. 39 Acne neonatorum and acne infantum are the earliest types. Acne vulgaris encompasses the subtypes comedonica, papulopustulosa and conglobata.…”

Section: Phenotypic Variationmentioning

confidence: 99%

Acne and hidradenitis suppurativa

2018

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Acne and hidradenitis suppurativa (HS) both centre on hair follicles. They often occur together as part of the acne tetrad, but are found in distinct localizations. Acne is primarily defined by the presence of comedones and inflammatory lesions. However, in HS the intertriginous localization and chronicity play equally important roles for the diagnosis to the inflammatory lesions. Genetics, bacteria, environmental factors and innate inflammation have all been found to play a role in acne and/or HS. Surprisingly, there is little overlap between the findings so far. The genetics of acne and HS are distinct, bacteria have not been shown convincingly to play a role in HS, and the important risk factors obesity and smoking in HS cannot be easily translated to acne. The one driving factor central to both diseases is innate inflammation, most strikingly involving interleukin-1. Hence the interleukin-1 family, as already shown in autoinflammatory conditions associated with acne, could represent attractive treatment targets.

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“…Acneiform dermatoses include a wide range of diseases which can occur in all age groups from infancy to elderly. They are so prevalent in all around the world that only AV rate has been reported to be 40-50 million/year in the USA and its prevalence has been estimated as 100% among adolescents in United Kingdom [2,3]. Acneiform dermatoses have also important economic burden on governments due to high prevalence [2,4].…”

Section: The Deinition Of 'Acne' and 'Acneiform'mentioning

confidence: 99%