Nonobstructive Hydronephrosis and Hydroureter Associated with Peritonitis

Makker, Sudesh P; Tucker, Arthur S.; Izant, Robert J.; Heymann, Walter

doi:10.1056/nejm197209142871104

Cited by 25 publications

(7 citation statements)

References 5 publications

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“…Several reports have focused on urologic complications caused by ingestion of a fish bone, most often a perivesical abscess or granuloma formation 2 . We found no previous reports of hydronephrosis caused by an intra‐abdominal abscess from cecal perforation by an ingested fish bone, although we did find a case of hydronephrosis caused by appendiceal abscess that had resulted from migration of an ingested metallic pin, 3 as well as 36 cases of hydronephrosis complicating peritonitis, appendicitis, or appendiceal abscess 4–10 …”

Section: Discussionmentioning

confidence: 67%

“…In the 38 cases of hydronephrosis caused by intra‐abdominal inflammation including the present case, treatment typically was drainage of any abscess present with or without bowel resection, or conservative therapy with antibiotics; 3–8 while only three patients required resection and reconstruction of an involved ureter because of severe adhesions or suspicion of cancer 9,10 . In most cases hydronephrosis resolved spontaneously after treatment for abscess.…”

Section: Discussionmentioning

confidence: 82%

“…In most cases hydronephrosis resolved spontaneously after treatment for abscess. They required a considerable period for complete resolution of the hydronephrotic changes following treatment of an abscess, as in the current case 4–7 . An impaired ureteral stream under such conditions is caused not only by ureteral compression by inflammatory masses or obstructive inflammatory changes in the ureter, but also by paralytic ureteritis occurring in response to extension of inflammation from the intra‐abdominal abscess 7,8 .…”

Section: Discussionmentioning

confidence: 88%

“…2 We found no previous reports of hydronephrosis caused by an intra-abdominal abscess from cecal perforation by an ingested fish bone, although we did find a case of hydronephrosis caused by appendiceal abscess that had resulted from migration of an ingested metallic pin, 3 as well as 36 cases of hydronephrosis complicating peritonitis, appendicitis, or appendiceal abscess. [4][5][6][7][8][9][10] In the 38 cases of hydronephrosis caused by intraabdominal inflammation including the present case, treatment typically was drainage of any abscess present with or without bowel resection, or conservative therapy with antibiotics; [3][4][5][6][7][8] while only three patients required resection and reconstruction of an involved ureter because of severe adhesions or suspicion of cancer. 9,10 In most cases hydronephrosis resolved spontaneously after treatment for abscess.…”

Section: Discussionmentioning

confidence: 99%

“…They required a considerable period for complete resolution of the hydronephrotic changes following treatment of an abscess, as in the current case. [4][5][6][7] An impaired ureteral stream under such conditions is caused not only by ureteral compression by inflammatory masses or obstructive inflammatory changes in the ureter, but also by paralytic ureteritis occurring in response to extension of inflammation from the intra-abdominal abscess. 7,8 This mural inflammation might require a considerable period for clearance of inflammatory cells and mediators in order to permit resumption of normal ureteral peristalsis.…”

Section: Discussionmentioning

confidence: 99%

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Hydronephrosis caused by intra‐abdominal abscess from cecal perforation by an ingested fish bone

et al. 2006

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Section: Discussionmentioning

confidence: 67%

Section: Discussionmentioning

confidence: 82%

Section: Discussionmentioning

confidence: 88%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Hydronephrosis caused by intra‐abdominal abscess from cecal perforation by an ingested fish bone

et al. 2006

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Gastrointestinal Infections

Marks

1985

Pediatric Infectious Diseases for the Practitioner

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Gastrointestinal infections are second only to respiratory infectious diseases in frequency and morbidity. A conservative estimate places the annual worldwide mortality from gastroenteritis-related infections at six million children. Although stomatitis and esophagitis are occasionally seen as distinct entities, the vast majority of gastrointestinal infections manifest as vomiting and/or diarrhea syndromes, commonly referred to as gastroenteritis. In many cases the term "enterocolitis" describes the pathologic and clinical features more accurately. EpidemiologyDiarrhea is most pronounced in communities where undernutrition, poor hygiene, and poorly developed community facilities for sewage and drinking water exist. In such communities, as recently described for Bangladesh, the peak incidence of diarrhea syndromes occurs between 2 and 11 months of age, and most infants in their first year of life can be expected to have seven episodes of gastroenteritis. 38 Expression and frequency of infection are related to decreased host resistance secondary to nutritional deficiencies, and increased spread referable to crowded living quarters, contaminated water and food, inadequate washing and toilet facilities, abundant flies, and inadequate refrigeration and cooking technology.Young infants are most prone to the dehydrating and debilitating effects of acute gastroenteritis and have the highest attack rates. Some organisms, such as Shigella, predominate in the summer and fall months, whereas others, such as rotavirus, are frequent in the winter months in temperate climates 53 and year-round in the tropics. 38 Campylobacter and enterotoxigenic E. coli are also very prevalent in such areas. 94 M. I. Marks, Pediatric Infectious Diseases

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