Chemical Biology 2012
DOI: 10.5772/34980
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Nitric Oxide Signaling During Senescence and Programmed Cell Death in Leaves

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“…Cytokinin induced the expression or activity of NR which caused a delayed leaf senescence. The involvement of receptor CYTOKININ-INDEPENDENT 1 (CKI-1) and ARABIDOPSIS RESPONSE REGULATOR (ARR) 2 were shown in this process (Mishina et al, 2007;Misra et al, 2012). It was also reported that overexpression of Hmp, an Escherichia coli NO dioxygenases (NOD) enzyme causing NO deficiency, in Arabidopsis after its activation, induced a senescence-like process which had many similarities with natural senescence (Mishina et al, 2007).…”
Section: -Pratap-singh-9780128187975mentioning
confidence: 97%
“…Cytokinin induced the expression or activity of NR which caused a delayed leaf senescence. The involvement of receptor CYTOKININ-INDEPENDENT 1 (CKI-1) and ARABIDOPSIS RESPONSE REGULATOR (ARR) 2 were shown in this process (Mishina et al, 2007;Misra et al, 2012). It was also reported that overexpression of Hmp, an Escherichia coli NO dioxygenases (NOD) enzyme causing NO deficiency, in Arabidopsis after its activation, induced a senescence-like process which had many similarities with natural senescence (Mishina et al, 2007).…”
Section: -Pratap-singh-9780128187975mentioning
confidence: 97%