2007
DOI: 10.1016/j.taap.2007.04.002
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Nicotine promotes cell proliferation via α7-nicotinic acetylcholine receptor and catecholamine-synthesizing enzymes-mediated pathway in human colon adenocarcinoma HT-29 cells

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Cited by 139 publications
(137 citation statements)
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“…As a particular case, previous studies on normal and cancer cells have suggested that nicotine, a major active component of cigarette smoke, can regulate the expression of catecholamine-synthesis pathway through the action on α7-nAChR, epinephrine production and ultimately epinephrine release and adrenergic receptors activation [6,13,14], Schemes 2 and 3. The α2-adrenoceptor is a transmembrane G protein-coupled receptor (GPCR) associated with the G i heterotrimeric G-protein.…”
Section: Introductionmentioning
confidence: 99%
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“…As a particular case, previous studies on normal and cancer cells have suggested that nicotine, a major active component of cigarette smoke, can regulate the expression of catecholamine-synthesis pathway through the action on α7-nAChR, epinephrine production and ultimately epinephrine release and adrenergic receptors activation [6,13,14], Schemes 2 and 3. The α2-adrenoceptor is a transmembrane G protein-coupled receptor (GPCR) associated with the G i heterotrimeric G-protein.…”
Section: Introductionmentioning
confidence: 99%
“…Studies suggested that smokers have a higher risk for developing colorectal cancer cells than non-smokers [6].…”
Section: Introductionmentioning
confidence: 99%
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