New Q Waves after Aortocoronary Bypass Surgery

Carotid Artery Stenosis Associated with Increased Mortality in Patients who Underwent Coronary Artery Bypass Grafting: A Single Center Experience

“…Other clinical outcomes analyzed were: cardiogenic shock; intra- and postoperative myocardial infarction – diagnosis confirmed by presence of two of the following factors: new Q waves on two or more ECG leads [12], CK-MB > 4 times the upper normal limit [13] or new segmental cardiomyopathy with akinesis on echocardiogram [14]; prolonged mechanical ventilation (>48h); prolonged use of vasopressors (>48h); sepsis; general infections (respiratory and urinary infection, endocarditis, surgical wound infection, and mediastinitis); respiratory infection (diagnosis based on chest X ray, leukocyte count, and compatible clinical findings); congestive heart failure; need for percutaneous transluminal coronary angioplasty; reoperation; conduction disorders requiring a temporary pacemaker; major bleeding (blood flow at the chest tubes over 10 mL/kg/h in one hour, 5 mL/kg/h in three consecutive hours, or 100 mL/h in four hours); pericarditis; fibrillation or atrial flutter.…”

Unspecific elevation of plasma troponin‐T and CK‐MB after coronary surgery

“…Furthermore, biochemical markers of myocardial injury are frequently used to evaluate the degree of myocardial injury when different treatment modalities and myocardial protective strategies are compared. On the other hand, it has been recognized that enzyme criteria can be unreliable because of unspecific elevation due to surgical tissue trauma, cardiopulmonary bypass (CPB), transient ischaemia during aortic cross-clamping and retransfusion of mediastinal shed blood (3)(4)(5)(6). The introduction of more cardiospecific biochemical markers such as troponins have not solved this problem, as they seem to be similarly influenced by measures such as retransfusion of mediastinal shed blood.…”

Exercise‐Attenuation of Q‐Waves in II, III, and aVF, and R‐Waves in V1 and V2 in a Patient with an Inferior Infarction and Anterior Wall Ischemia

“…This can be pictured as an electrical “silence,”“inertia,” or “stunning” of a strategically poised myocardial territory opposite to a region with an old MI causing the transient masking of the Q‐ and R‐waves. Finally, in this last category of electrical scar‐ischemia interactions belongs the inverse to the presented herein ECG phenomenon, that of the de novo appearance of permanent Q‐waves (from an old MI) and which were masked previously engendered by the improved perfusion of the contralateral to the old MI myocardial territory following aortocoronary bypass grafting 8 …”