Neurophysiologie des Koronarschmerzes

Droste, Conrad

doi:10.1007/978-3-642-69451-6_4

Cited by 5 publications

(1 citation statement)

References 73 publications

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“…Further research should also consider the role of cognitive processes that define the presence or absence of ischemic pain [9]. Basic biochemical (e.g., arachidonic acid metabolites) and mechanical factors involved in coronary pain [7,8], also remain unclear.…”

Section: Discussionmentioning

confidence: 99%

A defective angina pectoris pain warning system: Experimental findings of ischemie and electrical pain test

Droste

Greenlee

Roskamm

1986

Pain

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Ischemic pain threshold and tolerance levels using the tourniquet pain technique and electrical cutaneous pain thresholds were measured in patients with asymptomatic ischemic heart disease. Thirty asymptomatic patients, who repeatedly exhibited no angina pectoris pain during the occurrence of exercise-induced coronary ischemia (greater than or equal to 0.1 mV ST segment depression in exercise ECG) were compared to 30 randomly selected symptomatic control patients. In a smaller patient group (6 symptomatic, 6 asymptomatic) the degree of forearm ischemia during the tourniquet test was determined non-invasively by monitoring transcutaneous pO2. Results indicated that asymptomatic patients needed significantly more time to reach pain threshold following occlusion of forearm blood flow and exhibited significantly lower tcpO2 values at threshold than symptomatic patients. Electrical pain thresholds were also elevated in the asymptomatic group. These findings indicate that the phenomenon of asymptomatic myocardial ischemia can be explained by an extracardiac pain modifying mechanism.

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Section: Discussionmentioning

confidence: 99%