Neonatal encephalopathy: An indicator of early sexual maturation in girls

Robertson, Charlene M.T.; Morrish, Donald W.; Wheler, G. H. T.; Grace, Michael

doi:10.1016/0887-8994(90)90042-y

Cited by 24 publications

(10 citation statements)

References 22 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…and 1 (Kappy & Ganong, 1994) and 23 and 1 (Bridges et al , 1994). There is a predisposition for the development of CPP in children with neurofibromatosis type 1 (2·4 -5%: Habiby et al , 1995Habiby et al , , 1997Cnossen et al , 1997;Carmi et al , 1999;Virdis et al ., 2000), in children with hydrocephalus (10 -11%: Abdolvahabi et al ., 2000;Brauner et al , 1987a;De Luca et al , 1985;Kaiser et al , 1989;Lopponen et al , 1996;Siddiqi et al , 1999;Tomono et al , 1983), in children with meningomyelocele (5-33%: Hunt, 1990;Meyer & Landau, 1984;Trollmann et al , 1996Trollmann et al , , 1998, in children with neonatal encephalopathy (4·3%: Robertson et al , 1990), and in children after low dose cranial irradiation (Brauner et al , 1984(Brauner et al , , 1999Ogilvy-Stuart et al , 1994). An association of Williams-Beuren syndrome with an increased frequency of CPP was recently reported (Scothorn & Butler, 1997;Cherniske et al , 1999;Partsch et al , 1999b).…”

Section: Figmentioning

confidence: 97%

See 1 more Smart Citation

Management and outcome of central precocious puberty

Partsch

Heger

Sippell

2002

Clinical Endocrinology

172

121

View full text Add to dashboard Cite

Section: Figmentioning

confidence: 97%

“…, 1983), in children with meningomyelocele (5–33%: Hunt, 1990; Meyer & Landau, 1984; Trollmann et al. , 1996, 1998), in children with neonatal encephalopathy (4·3%: Robertson et al. , 1990), and in children after low dose cranial irradiation (Brauner et al.…”

Section: Aetiology Incidence and Clinical Course Of Precocious Pubertymentioning

confidence: 99%

Management and outcome of central precocious puberty

Partsch

Heger

Sippell

2002

Clinical Endocrinology

172

121

View full text Add to dashboard Cite

“…There is a predisposition for the development of CPP in children with neurofibromatosis type 1 [40,41], in children with hydrocephalus [42,43], meningomyelocele [44,45], craniopharyngioma [46], neonatal encephalopathy [47], in children with chromosomal aberrations [48][49][50], and in children who have undergone low dose cranial irradiation [51,52]. An association of Williams-Beuren syndrome with an increased frequency of CPP was recently reported [53][54][55][56][57].…”

Section: Normal Pubertymentioning

confidence: 98%

Gonadotropin-Releasing Hormone Analogue Treatment for Precocious Puberty

Heger

Sippell

Partsch

2005

Endocrine Development

View full text Add to dashboard Cite

Central precocious puberty (CPP) is the premature onset of puberty due to a precocious activation of gonadotropin-releasing hormone (GnRH) neurons in the hypothalamus. This condition results in accelerated development of secondary sex characteristics, accelerated bone maturation, impaired final height with disproportioned body appearance and can have a disturbing impact on the psychosocial behavior of children suffering from CPP. It is therefore necessary to assess the hormonal status of children who show pubertal signs before the age 8 years in girls and 9 years in boys. The indication for treatment should be made after evaluating pubertal progression, progression of bone age maturation and final height prognosis, development of reproductive function, and psychosocial adjustment and well-being. This paper summarizes the experience of GnRH agonist treatment, which is momentarily the treatment of choice for central precocious puberty in children.

show abstract

“…4 Our patient had GH deficiency as a result of neonatal asphyxia and later developed central precocious puberty, with hormonal conditions of increased circulating levels of E2 and decreased levels of IGF-I. The ultrasonographic appearance of the ovaries indicated that IGF-I may not be essential for gonadotropin secretion and subsequent follicular development in the ovary, although GH replacement therapy may ameliorate fertility in some cases of GH deficiency.…”

Section: Discussionmentioning

confidence: 83%