Negative pressure pulmonary edema following thyroidectomy

Sharma, Manohar; Beckett, Neal; Gormley, Paul E.

doi:10.1007/bf03020501

Cited by 14 publications

(7 citation statements)

References 3 publications

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“…[1011] An entirely different population carries high NPPE risk which consist of the patients who are obese and patients who have obstructive sleep apnea syndrome, mediastinal mass, and upper respiratory tract obstruction. [121314] Depending on the clinical circumstance, if diagnosed early, many patients do not need aggressive therapies but some patients will need invasive or non-invasive mechanical ventilation. Patients usually respond to well fluid restriction, diuretics and corticosteroids.…”

Section: Discussionmentioning

confidence: 99%

Negative pressure pulmonary edema following septoplasty surgery triggering acute subendocardial myocardial in farction

et al. 2014

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Negative pressure pulmonary edema (NPPE) is defined as fluid transudation into the pulmonary interstitium which occurs as a result of elevated negative intrathoracic pressure caused by the upper respiratory tract obstruction and strong inspiratory effort. NPPE is usually seen during emergence from general anesthesia in the early post-operative period especially after upper respiratory tract surgery. We present a case of a 37-year-old male patient who underwent septoplasty operation and developed NPPE which could not diagnosed and progressed to acute subendocardial myocardial infarction.

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Section: Discussionmentioning

confidence: 99%

Negative pressure pulmonary edema following septoplasty surgery triggering acute subendocardial myocardial in farction

et al. 2014

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“…Initially 100 per cent oxygen can be given by a facemask. [10,11,12] If the PO 2 cannot be maintained at 60 mmHg or if there is progressive hypercapnia the patient should be intubated and mechanically ventilated. Application of PEEP of 5-20 cm of H 2 O is beneficial in these patients as it can decrease the preload and after load thereby improving cardiac function.…”

Section: Discussionmentioning

confidence: 99%

“…Fursemide is commonly used as it is venodilator and can reduce the preload rapidly. [12] Morphine also acts as transient venodilator and helps to reduce the preload in addition to relieving dyspnea and anxiety. These measures can decrease stress leading to reduction of catecholamine levels, tachycardia and ventricular afterload in pulmonary oedema.…”

Section: Discussionmentioning

confidence: 99%

Negative Pressure Pulmonary Oedema After Routine Elective Surgery

Singh¹,

Singh²,

S³

2014

International Journal of Medical and Dental Sciences

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Negative Pressure Pulmonary Oedema (NPPE) is known to occur in healthy subjects in the early post anaesthesia period, in the absence of fluid overload or left ventricular dysfunction. This type of non cardiogenic pulmonary oedema is also reported in literature following upper airway obstruction. We report two such cases of negative pressure pulmonary edema. Both the patients developed NPPE during postanaesthesia period due to persistent laryngospasm leading to upper airway obstruction. Both the patients were treated with diuretics, bronchodilators & antibiotics in intensive care unit. Within 24 hours the coarse crepitations disappeared and the patients were weaned off the ventilator. Awareness, early recognition and prompt treatment of negative pressure pulmonary oedema could be life saving.

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“…An extremely rare case of NPPE was described, induced by direct suction of endotracheal tube adapter (23). During postanesthesia, NPPE is observed more often among obese patients, patients with short neck or with sleep apnea (24), and patients who underwent ear, nose, and throat surgery (6,25).…”

Section: Discussionmentioning

confidence: 99%

Pulmonary edema and hemorrhage as complications of acute airway obstruction following anesthesia

Marchertienė¹,

Macas²,

Karbonskienė³

2008

Medicina

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Airway obstruction is a quite common complication while its conditioned pulmonary edema – rare. Causes associated with anesthesia are various. Forced inspiratory efforts against an obstructed upper airway generate peak negative intrathoracic pressure. This may cause pulmonary edema and in some cases pulmonary hemorrhage. Last-mentioned is extremely rare. Pulmonary edema may arise soon after airway obstruction as well as later, after some hours. Damage of bronchi is found seldom during bronchoscopy in case of pulmonary hemorrhage, while more often alveolar damage is observed due to alveolar membrane damage. Hemorrhage is conditioned by hydrostatic pressure level, level of hypoxia, damage to bronchi or alveoli (disruption of alveolar membrane). Early diagnosis of negative-pressure pulmonary edema or pulmonary hemorrhage is very important, because this affects postoperative morbidity and mortality of the patients. Two cases of pulmonary edema and hemorrhage after upper airway obstruction as well as literature overview are presented in this article. Pulmonary hemorrhage developed during anesthesia with ketamine, conditioned by increment of hydrostatic pressure, hypoxia, and effects of ketamine on hemodynamics.

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Negative pressure pulmonary edema following thyroidectomy

Cited by 14 publications

References 3 publications

Negative pressure pulmonary edema following septoplasty surgery triggering acute subendocardial myocardial in farction

Negative pressure pulmonary edema following septoplasty surgery triggering acute subendocardial myocardial in farction

Negative Pressure Pulmonary Oedema After Routine Elective Surgery

Pulmonary edema and hemorrhage as complications of acute airway obstruction following anesthesia

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