Nature and nurture: Genetic contributions to measures of the family environment.

Plomin, Robert; Reiss, David; Hetherington, E. Mavis; Howe, George W.

doi:10.1037/0012-1649.30.1.32

Cited by 225 publications

(227 citation statements)

References 30 publications

(40 reference statements)

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“…These composites are somewhat different from those previously published for the NEAD project (e.g., Plomin et al, 1994;Reiss et al, 2000). In order to create composites that would be valid and identical for both projects, we factor analyzed the measures that were available for both projects using the data from the TM sample.…”

Section: Methodscontrasting

confidence: 94%

Genetic and Environmental Influences on Mothering of Adolescents: A Comparison of Two Samples.

Neiderhiser¹,

Reiss²,

Pedersen³

et al. 2004

Developmental Psychology

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This study examined 2 samples of adolescents and mothers using a child-based design (Nonshared Environment in Adolescent Development [NEAD] project, N = 395 families) and a parent-based design (Twin Moms [TM] project, N = 236 twin family pairs) to compare genetic and environmental influences on mothering. For both samples, the same measures of positivity, negativity, control, and monitoring were used. The use of matched child-based and parent-based samples enabled passive and nonpassive genotype-environment (GE) correlations to be approximated, providing information about process. Passive GE correlations were suggested for mother's positivity and monitoring. For mother's negativity and control, primarily nonpassive GE correlations were suggested. In several cases, both types of GE correlation were indicated. Finally, observer ratings of negativity and monitoring were influenced only by environmental factors.

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Section: Methodscontrasting

confidence: 94%

Genetic and Environmental Influences on Mothering of Adolescents: A Comparison of Two Samples.

Neiderhiser¹,

Reiss²,

Pedersen³

et al. 2004

Developmental Psychology

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192

295

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“…Children who are risk for depression may follow a stochastic pathway: (1) genes that predispose to anxiety also predispose to the occurrence of certain depressogenic life events, that, in the face of a genetic diathesis to anxiety, lead to depression (127). Finally, studies of twins reared apart show that dimensions of maternal behavior such as parental warmth are actually heritable, being induced by the behavior of the twin offspring (12).…”

Section: Interactions Between Genes and Environmentmentioning

confidence: 99%

“…However, unless twin studies are combined with adoption studies (i.e., comparison of twins adopted away to different parents), it is difficult to definitively differentiate shared environmental from genetic effects. For example, in a twins-adopted away design, components of maternal behavior previously considered "environment" were explained by genetic concordance of MZ twins eliciting similar maternal responses from unrelated mothers (12). A second limitation is that MZ vs. DZ concordance may be differentially affected by shared or distinct perinatal experiences (13).…”

Section: Twin Studies (Seementioning

confidence: 99%

Familial Transmission of Suicidal Behavior

Brent¹,

Melhem²

2008

Psychiatric Clinics of North America

257

199

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Suicide and suicidal behavior is highly familial, and appears to be familially transmitted independently from the familial transmission of psychiatric disorder per se (1). Adoption, twin, and family studies support the view that the etiology of the familial transmission of suicidal behavior is at least in part genetic, and may be mediated by the transmission of intermediate phenotypes, such as impulsive aggression. In addition, there may be environmental causes for familial transmission, including imitation, and the intergenerational transmission of family adversity. In this review, we cover the evidence supporting the familial transmission of suicidal behavior, possible genetic and environmental explanations for this phenomenom, describe putative intermediate phenotypes, and discuss the contributory roles of early child-rearing and concurrent familial environmental stressors to suicidal risk. A better understanding of the mechanisms for the familial transmission of suicidal behavior can help to shed light on etiology, identify individuals at high risk for the development of incident suicidal behavior, and frame targets for intervention and prevention.Adoption studies (see Table 1) Three adoption studies have been conducted, all using the same Danish adoption registry. Kety et al., in a study designed to examine the genetics of schizophrenia and mood disorders, found a non-significant trend towards higher concordance for suicide in biological, compared to adoptive relatives of adoptees who committed suicide (2). Subsequently, a second study compared the rates of suicide among the biological and adoptive relatives of adoptees who committed suicide vs. biological and adoptive relatives of a matched living adoptee control group in Denmark (3). This study found a six-fold higher rate of suicide in the biological relatives of the suicide vs. those of the control adoptees, and an absence of suicide among the adopted relatives of the suicide vs. control adoptees supporting a genetic rather than environmental etiology (see Table 1). The rate of suicide was higher in the biologic relatives of suicide adoptees regardless of whether the adoptees were psychiatric patients or not. However, it was not possible to determine if the genetic liability to suicide was attributable to the transmission of major psychiatric disorders or to a suicide diathesis per se.In a third adoption study using this registry, a comparison of biological and adoptive relatives of adult adoptees with mood disorder and matched unaffected adoptees were examined (4), revealed a 15-fold increase in suicide among the biological relatives of the mood-disordered adoptees vs. those of the unaffected adoptees (4). This finding supports the role of mood disorder in the genetics of suicide. However, the greatest increased risk for suicidal behavior was found in the relatives of those probands with "affect reaction," a diagnosis akin to Corresponding Author: Dr. David Brent, BrentDA@upmc.edu. Publisher's Disclaimer: This is a PDF file of an unedited manuscript th...

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“…In general, twin studies have focused largely on broad dimensions of parenting, providing evidence of significant genetic contributions in relation to both positive and negative aspects of parental affect (e.g., warmth, negativity), but there is less support for the role of genes with regard to parental control (Braungart, 1994;Elkins et al, 1997;Goodman and Stevenson, 1991;Plomin et al, 1989Plomin et al, , 1994Rowe, 1981). Although these findings are not entirely consistent across studies (Reiss et al, 2000) or sources of reports within studies (Deater-Deckard, 2000), it does appear clear that affective dimensions of parenting yield greater genetic sources of variation than nonaffective control-related dimensions (e.g., see Lichtenstein et al, 2003;Losoya et al, 1997).…”

mentioning

confidence: 99%

Examining the Etiology of Associations Between Perceived Parenting and Adolescents' Alcohol Use: Common Genetic and/or Environmental Liabilities?

Latendresse¹,

Rose²,

Viken³

et al. 2010

J. Stud. Alcohol Drugs

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ABSTRACT. Objective: Although twin studies yield consistent evidence of heritability in the frequency of adolescent alcohol use, parallel findings on parenting are more equivocal, supporting the role of genes in affective parenting but not as supportive in relation to parental control. The extent to which these patterns generalize to more nuanced forms of parenting is less clear. Furthermore, despite evidence linking parents' socialization practices with adolescents' alcohol-use behaviors, this study is the first attempt to determine the sources of this covariation. Method: The present study used epidemiological data from 4,729 adolescent twins (2,329 females) to examine the nature of associations between their perceptions of parenting at age 12 and frequencies of alcohol use at age 14. Univariate analyses assessed the relative contributions of genetic and environmental influences on variability within six domains of parenting. Among those displaying consistent evidence of heritability, bivariate models were used to explore sources of covariation with drinking frequency. Results: Univariate models suggested both genetic and environmental sources of variability across parenting phenotypes, including sex-specific sources of genetic liability within one dimension of parenting. However, despite evidence for heritability, bivariate analyses indicated that the covariation between alcohol use and perceptions of parental knowledge and warmth were entirely mediated through shared environmental pathways. Conclusions: This study elucidates genetic and environmental sources of variability within individual parenting behaviors and characterizes the etiological nature of the association between parental socialization and adolescent alcohol use. The identification of specific and modifiable socialization practices will be crucial for the future development of parent-based prevention/intervention strategies. (J. Stud. Alcohol Drugs, 71, 313-325, 2010)

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Nature and nurture: Genetic contributions to measures of the family environment.

Cited by 225 publications

References 30 publications

Genetic and Environmental Influences on Mothering of Adolescents: A Comparison of Two Samples.

Genetic and Environmental Influences on Mothering of Adolescents: A Comparison of Two Samples.

Familial Transmission of Suicidal Behavior

Examining the Etiology of Associations Between Perceived Parenting and Adolescents' Alcohol Use: Common Genetic and/or Environmental Liabilities?

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