“…superoxide dismutase or heat shock proteins), or inhibit the fusion of phagosome and lysosome [42]. The recognition of MAC by macrophages mainly acts through toll-like receptor 2 (TLR2) [43]; this leads to the production of pro-inflammatory cytokines, such as interleukins IL-1β, IL-12, IL-18, tumour necrosis factor α (TNFα), as well as chemokines such as the C-X-C motif chemokine 10 (CXCL-10) [42]. Chemokines and TNFα cause taxis of inflammatory cells such as lymphocytes, macrophages and dendritic cells, to the inflammation site.…”