Muscle performance and ankle joint mobility in long-term patients with diabetes

Abstract

Background: Long-term patients with diabetes and peripheral neuropathy show altered foot biomechanics and abnormal foot loading. This study aimed at assessing muscle performance and ankle mobility in such patients under controlled conditions.

Assessment of lower leg muscle force distribution during isometric ankle dorsi and plantar flexion in patients with diabetes: a preliminary study

“…We need to note that the current study examined the muscle force contribution of diabetic neuropathic patients; hence the observed difference between the groups is attributed to the combined effect of neuropathy and diabetes. Giacomozzi et al (2008), when examining groups of diabetic and diabetic neuropathic patients against healthy participants, attributed the overall decrease in the ankle moment to muscle atrophy as a result of muscle tissue glycation and damage as a result of impaired nerve conduction. These differences together with alterations of cartilages, ligaments and tendons as a result of glycation (Giacomozzi, D'Ambrogi, Uccioli, & Macellari, 2005;Wrobel & Najafi, 2010) could explain the deterioration in the muscle force contribution during isometric plantar and dorsi flexion contractions.…”

Avaliação de um programa para prevenção de úlceras neuropáticas em portadores de diabetes

“…An otherwise simple lesion can lead to functional losses 2,3 and culminate in loss of the limb or even death 4 . Several factors are involved in the development of foot ulcers in diabetic patients: neuropathy, peripheral vascular disease, limitation of joint motion, trophic skin disorders and abnormal distribution of mechanical forces in the feet [5][6][7] . Among them, the most important etiologic factor is diabetic peripheral neuropathy 5,[8][9][10] .…”

Padrões dinâmicos de recrutamento muscular por EMG de alta densidade de diabéticos em diferentes níveis de severidade de neuropatia classificados por um modelo linguistico fuzzy

“…Todas essas alterações provavelmente são decorrentes da glicosilação progressiva do colágeno, e prejudicam a adaptação fisiológica do pé ao solo durante a marcha. Em outro estudo, Giacomozzi et al (2008) observaram uma diminuição da amplitude de movimento do tornozelo, relacionada a alterações estruturais da cartilagem e da cápsula articular do complexo tornozelo-pé. Os mesmos autores identificaram uma diminuição do torque isocinético do tornozelo de diabéticos neuropatas, sendo que a dorsiflexão apresentou maior comprometimento do que a flexão plantar.…”