Multiple pulp stones and shortened roots of unknown etiology

Parekh, Susan; Kyriazidou, Anna; Bloch‐Zupan, Agnès; Roberts, Graham

doi:10.1016/j.tripleo.2005.11.003

Cited by 25 publications

(16 citation statements)

References 13 publications

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“…Both category of pulp stones may element eloquently occlude the canals and may be present at a curve which may baffle with the root canal treatment. 4 Kronfeld and Boyle 5 classified pulp stones histologically into ''true'' or ''false'' types. True pulp stones are much more aberrant in pattern and are lined by odontoblasts.…”

Section: Introductionmentioning

confidence: 99%

“…6 The pulp calcifications noticeably ensue all through the dentition in patients with systemic or genetic conditions like dentin dysplasia and dentinogenesis imperfecta. 4 The conditions like hypercalcaemia, gout, and renal calcifications that are noted secondary to the calcium metabolism have been observed as the pre-disposing factors for pulp stone formation. Pulpal calcification has been frequently ascertained in patients with end-stage renal disease (ESRD) and transplanted patients, and a positive correlation between the chronicity of the renal disease and the pulpal obliteration has been noticed in the premolar and molar teeth of such patients.…”

Section: Introductionmentioning

confidence: 99%

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Prevalence of and relationship between pulp and renal stones: A radiographic study

Patil

2015

Journal of Oral Biology and Craniofacial Research

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Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Prevalence of and relationship between pulp and renal stones: A radiographic study

Patil

2015

Journal of Oral Biology and Craniofacial Research

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“…The pulp calcifications in patients with systemic or genetic diseases such as dentin dysplasia and dentinogenesis imperfecta usually occur throughout the dentition [4]. The conditions which are secondary to the calcium metabolism, like hypercalcaemia, gout and renal lithiasis have been noted as the pre-disposing factors for the pulpal calcification.…”

mentioning

confidence: 99%

Pulp Stone, Haemodialysis, End-stage Renal Disease, Carotid Atherosclerosis

Patil

Sinha²

2013

JCDR

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Objectives: The aim of this study was to determine the relation ship between the presence of pulp calcification and carotid artery calcification on the dental panoramic radiographs in End Stage Renal Disease (ESRD) patients who were on haemodialysis.Methods: A total of 112 End Stage Renal Disease (ESRD) pati ents on who were haemodialysis participated in this study. The periapical and the panoramic radiographs for all the patients were evaluated for the presence or absence of the narrowing of the dental pulps and for pulp stones in the pulp chambers and the pulp canals. The panoramic radiographs were also evaluated to determine the carotid calcification.

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“…Over time, several case reports have documented evidence of a possible additional type of DD or a new, non‐syndromic dentin defect displaying generalized pulp stones in clinically normal, single‐rooted teeth and premolars, situated at the midroot level, with the roots bulging around the stones. Various names were given to these delineated entities, such as DD‐III or focal odontoblastic dysplasia , generalized focal pulpal calcification , or an atypical variation of O'Carroll's DD‐I subtype d .…”

Section: Classification Of Hereditary Dentin Disordersmentioning

confidence: 99%

Dentin disorders: anomalies of dentin formation and structure

Coster¹

2009

Endodontic Topics

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Dentin defects represent a cluster of relatively rare heritable disorders affecting the formation and mineralization of dentin. Major clinical features include loss of enamel with extreme wear of exposed dentin, opalescent tooth discoloration, short roots with pulpal calcification and increased tooth mobility, and frequent periapical radiolucencies. Historically, these inherited conditions have been classified by Shields et al. (1) as either dentinogenesis imperfecta (DGI) types I, II and III, or dentin dysplasia (DD) types I and II. DGI type I is now universally designated as osteogenesis imperfecta with dentinogenesis imperfecta (OI/DGI) and is caused by type I collagen mutations. Among the genes expressing the non‐collagenous proteins in dentin, only the DSPP gene has been implicated in the etiology of DD type II and all DGI types, suggesting that heritable dentin defects can be viewed as a continuum rather than as distinct disease entities. Dental treatment strategies should aim to preserve the affected dentitions by protecting teeth from wear, restoring them to compliance with the specific structural characteristics of the defective dentin, and anticipating early tooth loss. Endodontic treatment of the affected teeth is very challenging because of the peculiar pulp morphology and most often has poor prognosis. If conventional therapy is not an option, periapical curettage and retrograde filling is a possible alternative, except in the case of teeth with short roots.

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Multiple pulp stones and shortened roots of unknown etiology

Cited by 25 publications

References 13 publications

Prevalence of and relationship between pulp and renal stones: A radiographic study

Prevalence of and relationship between pulp and renal stones: A radiographic study

Pulp Stone, Haemodialysis, End-stage Renal Disease, Carotid Atherosclerosis

Dentin disorders: anomalies of dentin formation and structure

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