Multiple hormonal responses to graded exercise in relation to physical training.

Hartley, L. Howard; Mason, J W; Hogan, Ryan; Jones, Leighton; Kotchen, T A; Mougey, Edward H.; Wherry, Frances E.; Pennington, Lee L.; Ricketts, Percy T.

doi:10.1152/jappl.1972.33.5.602

Cited by 373 publications

(97 citation statements)

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“…On the other hand, alpha adrenergic blockade (unopposed beta stimulation) produced a significant elevation ofplasma free fatty acids both before and after exercise. These findings are consistent with earlier studies (36) and may account for the decrease iii glucose uptake observed during phentolamine infusion in the pre-exercise control period.…”

supporting

confidence: 93%

Adrenergic blockade alters glucose kinetics during exercise in insulin-dependent diabetics.

Simonson¹,

Koivisto²,

Sherwin³

et al. 1984

J. Clin. Invest.

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supporting

confidence: 93%

Adrenergic blockade alters glucose kinetics during exercise in insulin-dependent diabetics.

Simonson¹,

Koivisto²,

Sherwin³

et al. 1984

J. Clin. Invest.

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“…Floras e col 26 verificaram por técnica de microneurografia, que a diminuição da atividade simpática do nervo peronero em hipertensos limítrofes, observada 60min após uma sessão de exercício físico, coincidia com a diminuição volume 69, (nº 3), 1997 da PA. A ativação do sistema de opióides peptídicos tem sido sugerida como um dos prováveis mecanismos responsáveis por essa atenuação da atividade nervosa simpática periférica 27 . Os mecanismos pelos quais o exercício crônico, a longo prazo, atenuam a hipertensão, também não estão esclarecidos, mas parece que, em parte, eles podem ser explicados por alguns comportamentos: 1) após um programa de TF, a redução da atividade nervosa simpática periférica contribui para a atenuação da PA, visto que, observa-se que os níveis de norepinefrina circulante 22,25,[28][29][30] estão diminuídos; 2) o aumento da excreção urinária de sódio e, conseqüentemente, a diminuição da atividade da renina plasmática 21,25 são respostas que também têm sido utilizadas para explicar a diminuição da PA após TF.…”

Section: Discussionunclassified

Efeito do treinamento físico de membros superiores aeróbio de curta duração no deficiente físico com hipertensão leve

Haddad¹,

Silva²,

Barretto³

et al. 1997

Arq. Bras. Cardiol.

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“…Insulin secretion is inhibited by increased sympathetic nervous system activity via a-adrenergic receptors, and plasma insulin concentrations decline to low levels (6,(23)(24)(25). This results in increased lipolysis in adipose tissue and increased hepatic glucose production (26,27).…”

Section: Ls Hortonmentioning

confidence: 99%

Role and Management of Exercise in Diabetes Mellitus

Horton

1988

Diabetes Care

111

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As more is understood about the physiology of exercise, in both normal and diabetic subjects, its role in the treatment of diabetes is becoming better defined. Whereas people with diabetes may derive many benefits from regular physical exercise, there are also several hazards that make exercise difficult to manage. In type I (insulin-dependent) diabetes, there are risks of hypoglycemia during or after exercise or of worsening metabolic control if insulin deficiency is present. Type II (non-insulin-dependent) diabetic patients treated with sulfonylureas are also at some increased risk of developing hypoglycemia during or after exercise, although this poses less of a problem than with insulin treatment. In individuals treated by diet alone, regulation of blood glucose during exercise is usually not a significant problem and exercise can be used as an adjunct to diet to achieve weight loss and improved insulin sensitivity. When obese type II diabetic patients are treated with very low calorie diets, adequate amounts of carbohydrate must be provided to ensure maintenance of normal muscle glycogen content, particularly if individuals wish to participate in highintensity exercise that places a heavy workload on specific muscle groups. On the other hand, moderateintensity exercise such as vigorous walking can be tolerated by individuals on very low calorie, carbohydrate-restricted diets after an appropriate period of adaptation. A number of strategies can be employed to avoid hypoglycemia in type I diabetic patients, and both type I and II diabetic patients should be examined carefully for long-term complications of their disease, which may be made worse by exercise. These considerations have led many diabetologists to consider exercise beneficial in the management of diabetes for

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Multiple hormonal responses to graded exercise in relation to physical training.

Cited by 373 publications

References 0 publications

Adrenergic blockade alters glucose kinetics during exercise in insulin-dependent diabetics.

Adrenergic blockade alters glucose kinetics during exercise in insulin-dependent diabetics.

Efeito do treinamento físico de membros superiores aeróbio de curta duração no deficiente físico com hipertensão leve

Role and Management of Exercise in Diabetes Mellitus

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