2012
DOI: 10.4103/1817-1745.106489
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Motor aphasia: A rare complication of scorpion sting

Abstract: Scorpion sting is common in villages, and is an important public health problem in India. The clinical symptoms of envenomation by scorpion sting are by sympathetic and parasympathetic stimulation, causing a variety of symptoms. The leading causes of death are cardiac dysfunction and pulmonary edema. We present herein a case of scorpion sting in a 9-year-old boy who developed pulmonary edema and gradually developed cytotoxic cerebral edema with infarct leading to motor aphasia with upper motor neuron facial pa… Show more

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Cited by 3 publications
(4 citation statements)
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“…There are more than 1500 different species of scorpions in the world and only about 50 of them are medically important to humans [ 1 ]. The most dangerous scorpions are found in South America, North Africa, South Africa, Middle East, and India [ 2 ].…”
Section: Introductionmentioning
confidence: 99%
“…There are more than 1500 different species of scorpions in the world and only about 50 of them are medically important to humans [ 1 ]. The most dangerous scorpions are found in South America, North Africa, South Africa, Middle East, and India [ 2 ].…”
Section: Introductionmentioning
confidence: 99%
“…14 Neurological complications are well-recognized manifestations of scorpion stings in Mexico, Brazil, Turkey, India, and Venezuela. 5,[7][8][9]15,16 Cerebral infarcts usually manifest more than 48 to 72 hours after the initial scorpion envenomation, 5,15 which was the case of our patient with suspected scorpion sting, who could have had the neurological manifestation as a result of both pathogenic pathways.…”
Section: Discussionmentioning
confidence: 99%
“…1 Neurotoxins contained in the venom derive their potency from activating voltage-gated sodium channels and inhibiting potassium channels, leading to a prolonged depolarization of excitable cells in the peripheral nervous system with subsequent neurotransmitter release. 2,5 Scorpion stings present a wide spectrum of clinical presentations, ranging from pain at the inoculation site, a burning sensation, and radiation to the rest of the affected limb, and generally are not associated with local signs of inflammation. Systemic clinical manifestations usually start with a cholinergic phase (abdominal pain, vomiting, sialorrhea, bronchorrhea, sinus bradycardia, miosis, and increased levels of amylase and glucose), which can be followed (or not) by an adrenergic phase (piloerection, mucocutaneous pallor, hypertension, sinus tachycardia, cardiac arrhythmias, acute lung injury, and cardiogenic shock).…”
Section: Introductionmentioning
confidence: 99%
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