“…Suggested mechanisms include anatomic malformations, such as long mesosalpinx, hydatid of Morgagni, hydrosalpinx, abnormalities in adjacent organs (ovarian and paraovarian masses, uterine enlargement by pregnancy or tumor and peri-tubal adhesions) physiologic disturbances in the normal peristalsis of the tube, hemodynamic alterations causing congestion and tortuosity of the mesosalpingeal veins, trauma or accidents, or previous tubal surgeries [3][4][5]7].…”