2016
DOI: 10.1016/j.lfs.2016.02.072
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Methamphetamine-induced dopaminergic toxicity prevented owing to the neuroprotective effects of salicylic acid

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Cited by 33 publications
(12 citation statements)
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“…Likewise, another biomolecule Icariin has also benefitted against mitochondrial fragmentation associated with Aβ 1-42 accumulation in 3 × Tg-AD neurons [109]. Similarly, Salicylic acid has currently reported to provide neuroprotection against mitochondrial dysfunction, oxidative stress and apoptosis in PD brain due to its antioxidant and anti-inflammatory effects on neuronal cell [110]. Further, potential protective effect of Succinobucol against mitochondrial metabolic impairment and oxidative stress induced by 3-NP has been reported in HD brain [111].…”
Section: Biomolecules Mediated Therapy For Altered Mitochondrial Dynamentioning
confidence: 98%
“…Likewise, another biomolecule Icariin has also benefitted against mitochondrial fragmentation associated with Aβ 1-42 accumulation in 3 × Tg-AD neurons [109]. Similarly, Salicylic acid has currently reported to provide neuroprotection against mitochondrial dysfunction, oxidative stress and apoptosis in PD brain due to its antioxidant and anti-inflammatory effects on neuronal cell [110]. Further, potential protective effect of Succinobucol against mitochondrial metabolic impairment and oxidative stress induced by 3-NP has been reported in HD brain [111].…”
Section: Biomolecules Mediated Therapy For Altered Mitochondrial Dynamentioning
confidence: 98%
“…Oxidative stress occurs primarily in mitochondria and leads to mitochondrial dysfunction by attacking mitochondria in the CNS ( Thrash-Williams et al, 2016 ). Accordingly, METH-induced mitochondrial damage may contribute to dopaminergic toxicity by enhancing susceptibility to oxidative stress and promoting the apoptosis of neural cells.…”
Section: Mechanisms Underlying Meth-induced Neurotoxicitymentioning
confidence: 99%
“…Aside from addiction, it may lead to cardiotoxicity, hepatotoxicity, nephrotoxicity, and even to retinal damage 1 – 4 . In the central nervous system (CNS), cocaine use may result in neurotoxicity, neuroinflammation 5 8 and neurodegeneration 9 , 10 . Evidence associating the harmful outcome of cocaine toxicity with oxidative stress (OS) has accumulated during the last decade, and it is believed to play a main role in the negative consequences of cocaine intake.…”
Section: Introductionmentioning
confidence: 99%