Metabolic syndrome components in Indians migrants with type 2 diabetes subjects. A matched comparative study

Foucan, Lydia; Deloumeaux, Jacqueline; Donnet, J.-P.; Bangou, J.; Larifla, Laurent; Messerchmitt, C.; Salmi, L.R.; Kangambega, P.

doi:10.1016/j.diabet.2007.01.001

Cited by 8 publications

(13 citation statements)

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“…The remainder were excluded as they were conducted only among Indians living in India or did not meet the eligibility criteria. The studies were conducted in Australia, 26 Singapore, 27 Canada 28 Guadeloupe, 29 the Netherlands, 30 the USA 31–33 and the UK 14,34 . The sample sizes in the studies ranged from 143 33 to to 11 624 participants 32 .…”

Section: Resultsmentioning

confidence: 99%

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Prevalence of obesity among migrant Asian Indians: a systematic review and meta-analysis

Fernandez

Everett

2011

International Journal of Evidence-Based Healthcare

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Based on the available evidence, the obesity indices among migrant Asian Indians are significantly greater when compared with the native population and those living in India, particularly among women. This is likely to contribute to the high levels of diabetes and coronary heart disease in this population. Culturally appropriate strategies to reduce obesity, particularly abdominal obesity, in this ethnic group are urgently needed.

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Section: Resultsmentioning

confidence: 99%

“…All 10 studies reported on the mean BMI of migrant Asian Indians which ranged from 24 31 to 33 kg/m 2 33 . Pooled data from eight studies 14,26–31,34 demonstrated that migrant Asian Indians had significantly higher BMI values when compared with other migrants and the native population (SMD 0.36; 95% CI 0.30, 0.41) (Fig. 1).…”

Section: Resultsmentioning

confidence: 99%

Prevalence of obesity among migrant Asian Indians: a systematic review and meta-analysis

Fernandez

Everett

2011

International Journal of Evidence-Based Healthcare

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“…12 The prevalence of MetS in diabetic Indian immigrants in the USA was 77 %. 13 A prevalence of MetS in a cross sectional survey of Italian Diabetic patients by the Metascreen writing Committee in 2006 was 69.5%. 14 The prevalence of MetS in diabetic Nepalese population as per NCEP/ATP III in the present study is comparable to its prevalence reported in the above studies.…”

Section: Discussionmentioning

confidence: 99%

Prevalence of metabolic syndrome in type 2 diabetes mellitus patients using NCEP/ATP III and IDF criteria in Nepal

2012

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Background: With increasing trends towards sedentary life style, obesity, diabetes mellitus, hypertension, and dyslipidaemia, the prevalence of metabolic syndrome (MetS) is rising in our country. In view of importance of diagnosis of MetS in day to day medical practice for early institution of life style therapies to reduce the atherosclerotic cardiovascular disease risk in susceptible population like in diabetes mellitus, this study has been undertaken to study the prevalence of metabolic syndrome in Nepalese diabetes by applying National Cholesterol Education Programme/Adult Treatment panel (NCEP/ATP) III and International Diabetes Federation (IDF) criteria.

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“…Given the interaction between prenatal and postnatal conditions in influencing risk and the rapid rate at which environmental, and particularly nutritional, conditions are changing throughout the world, the fetal programming hypothesis has particularly worrisome public health implications Gluckman et al, In press). Studies of migrant populations from developing to developed countries indicate that a dramatic shift in the nutritional environment between gestation, childhood, and adult life may underlie the elevated risk for heart disease and diabetes that such populations face (Barnett et al, 2006;Beaulieu et al, 2007;Candib, 2007;Daryani et al, 2005;Dwivedi, Agarwal, Suthar, and Dwivedi, 2004;Foucan et al, 2006;Lob-Corzilius, 2007;Misra and Misra, 2003;Misra and Vikram, 2004;Misra, Endemann, and Ayer, 2005;Pousada et al, 2006;Schwingel et al, 2007;Trayhurn, 2005;Tull, Thurland, and LaPorte, 2005). Rapid nutritional transitions in the developing world may be accompanied by similar increase in chronic disease burden, if the fetal programming principle holds true.…”

Section: Fetal Programming In Human Epidemiologymentioning

confidence: 99%

Fetal programming and fetal psychology

Ellison

2010

Infant and Child Development

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The introduction of the "fetal programming hypothesis," first in epidemiology, subsequently in a broad range of disciplines concerned with developmental biology, has generated new interest in phenotypic plasticity, the mechanisms that govern it, and its place in evolutionary biology. A number of epidemiological studies link small size at birth, assumed to be a consequence of constrained prenatal energy availability, with adverse effects on the risk of chronic diseases later in life. The cluster of chronic diseases associated with the metabolic syndrome and alterations of glucose metabolism are particularly implicated. Recent evidence suggests that epigenetic modification of gene expression affecting the hypothalamic-pituitary-adrenal (HPA) axis may be involved in these effects. In animal studies epigenetic alteration of HPA axis activity and responsiveness is associated with changes in adult behavior and stress responsiveness.The potential for similar effects to contribute to psychological and psychiatric outcomes has been explored in a number of contexts, including famine exposure, observed covariance with birth weight, and prenatal dexamethasone treatment of fetuses at risk of congenital adrenal hyperplasia. While fetal programming effects have now been widely demonstrated across species and human populations, the adaptive significance of these effects is still a matter of debate.Life is integrated, cumulative, and continuous, not episodic, static, or discrete.Behavioral scientists rarely forget this, but biologists often do. Developmental biology is the one domain of the life sciences where the organism as a progressively unfolding phenomenon is a central concept. The reemergence of developmental biology as a vigorous discipline, intersecting in important ways with genetics (Badayev, 2008), evolution (West-Eberhard, 2003), and epidemiology (Barker, 1994;Gluckman and Hanson, 2006;Kuh, Ben-Shlomo, Lynch, Hallqvist, and Power, 2003), has injected new energy and new ideas into all those fields. Within epidemiology a seminal impact of this new attention to developmental biology has been in the formulation of the "fetal programming hypothesis," also known as the "fetal origins hypothesis," or, more generally, as the "developmental origins of health and disease" (DOHaD). Simply put, this hypothesis suggests that conditions very early in development, even in utero, can leave lasting imprints of an organism's physiology, imprints that may affect susceptibility to diseases with onsets that may occur many decades later (Barker, Eriksson, Forsen, and Osmond, 2002;.The concept of fetal programming is, of course, not new. Behavioral endocrinologists and neuroscientists, for example, have long recognized "organizational effects" of prenatal androgen hormones in "programming" certain aspects of reproductive axis function and reproductive behavior that emerges later in an animal's life (Nelson, 2005). "Critical periods" in development, including fetal development, are familiar

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Metabolic syndrome components in Indians migrants with type 2 diabetes subjects. A matched comparative study

Cited by 8 publications

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Prevalence of obesity among migrant Asian Indians: a systematic review and meta-analysis

Prevalence of obesity among migrant Asian Indians: a systematic review and meta-analysis

Prevalence of metabolic syndrome in type 2 diabetes mellitus patients using NCEP/ATP III and IDF criteria in Nepal

Fetal programming and fetal psychology

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