Metabolic Maladies in New Zealand Maoris

Prior, Ian A.; Rose, B.; Davidson, Flora

doi:10.1136/bmj.1.5390.1065

Cited by 48 publications

(18 citation statements)

References 8 publications

(3 reference statements)

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“…The Smith et al (1985) article also left open the possibility of a behavioral explanation at the time Caldararo wrote the article as did work on Japanese populations, while Thomson & Shaw (2002) proposed specific dietary additives in traditional Maori diet that might explain lower rates among Maori eating this diet. This conclusion seems at odds with data published a half century ago by Prior et al (1964).…”

Section: Introductionmentioning

confidence: 71%

“…The affects of diets on fecal water is the focus of work and some evidence has shown that change in pH can promote CRC. Since there is evidence of an increase in consumption of soft drinks among Polynesians and specifically Maori, comparing reports from nearly 50 years ago (Prior et al, 1964) with current levels reported by the New Zealand Ministry of Health (25 October 2007 from diabetes surveys, New Zealand Ministry of Health, 2007). This might explain some of the increase of CRC in the lower socio-economic group of Maori females reported by Maori Health.…”

Section: Discussionmentioning

confidence: 99%

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The Maori, Behavior, Modern Diets and Colorectal Cancers

Caldararo

2012

Asian Pacific Journal of Cancer Prevention

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Section: Introductionmentioning

confidence: 71%

Section: Discussionmentioning

confidence: 99%

The Maori, Behavior, Modern Diets and Colorectal Cancers

Caldararo

2012

Asian Pacific Journal of Cancer Prevention

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“…Earlier work described the link between "westernisation" and metabolic maladies of the New Zealand Maori and the serious implications it has for a disease complex including hypertension, coronary artery disease, obesity, diabetes, hyperuricaemia, gout, and, as a complication, renal failure [1,2]. A longitudinal survey of gout in the Tokelauans has shown a relative risk of gout of 9 in the migrants compared with the non-migrants [38].…”

Section: Discussionmentioning

confidence: 99%

“…The high prevalence rates of Type 2 (non-insulin-dependent) diabetes, gout, hypertension and coronary heart disease in some groups of Polynesians has initiated a series of epidemiological studies in the Pacific [1][2][3][4][5][6][7][8][9][10]. These studies suggest that Polynesians, and, to a varying degree, Melanesians and Micronesians [11,121, have a genetic predisposition to a disease complex of obesity, hypertension, coronary artery disease, gout and diabetes.…”

mentioning

confidence: 99%

Type 2 (non-insulin-dependent) diabetes mellitus, migration and westernisation: The Tokelau Island Migrant study

Østbye

Welby²,

Prior³

et al. 1989

Diabetologia

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Summary. The migration of Tokelauans from a traditional atoll in the Pacific to urban New Zealand is associated with an increased prevalence and incidence of Type 2 (non-insulindependent) diabetes mellitus over the period [1968][1969][1970][1971][1972][1973][1974][1975][1976][1977][1978][1979][1980][1981][1982]. During the same period, a lesser but definite increase is seen among non-migrants in Tokelau. The age standardised prevalence rates rose from 7.5 and 11.7 to 10.8 and 19.9 per 100 respectively in the male and female migrants compared with an increase from 3.0 and 8.7 to 7.0 and 14.3 per 100 in the nonmigrant males and females respectively. The incidence of diabetes is shown to be consistently higher in the migrants compared to the non-migrants giving relative risks of 1.5 in males and 1.9 in females. The factors most likely contributing to this difference, are changes to a higher calorie, high protein diet, higher alcohol consumption, a greater weight gain and altered levels of physical activity in the migrants. A number of populations in the Pacific have been shown to have a low rate of diabetes in their traditional setting, but may have a genetic predisposition for diabetes which responds to factors in the urban industrialised environment and life-style. The social and economic changes taking place in Tokelau are also clearly increasing the risk of diabetes. To reverse these trends and prevent the development of complications of Type 2 diabetes, it will be important to institute preventive programmes and to follow up the population in both environments for long-term outcomes, including mortality.

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“…26 Prior et al found that obese Maori had higher SUA than those who were not obese. 9 Burch et al reported that SUA was higher in larger individuals whether judged by height, weight, or body surface. 8 Such measurements and estimates in our population were tested for correlation with the SUA.…”

Section: Sua and Other Factorsmentioning

confidence: 99%