Meningioma of the scapula

Llauger, Jaume; Aixut, Sònia; Cañete, Noemí; Palmer, J.; Sola, Montserrat Martínez de; Bagué, Sílvia

doi:10.1007/s00256-007-0366-7

Cited by 8 publications

(9 citation statements)

References 7 publications

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“…Meningiomas are the most common benign tumors of the brain, representing 20% to 30% of all intracranial tumors. [2][3][4] Local invasion and distant metastasis of intracranial meningiomas have been described in the medical literature, 3,[5][6][7][8][9][10][11][12] but are rare. Access to the blood is thought to occur through the system of dural venous sinuses.…”

Section: Metastasis Of a Histologically Benign-appearing Meningioma T...mentioning

confidence: 99%

“…This suggests a spread of hematopoietic tumor cells from the PMGCT to blood, bone marrow, and/or extramedullary sites. 12 The median time for the development of hematologic neoplasia associated with PMGCT is 6 months (range, 0 to 47 months). 3 The clinical course of the hematologic neoplasia is very aggressive with a median survival of 5 months (range, 0 to 16 months) after diagnosis.…”

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confidence: 99%

“…3 Hematologic malignancies occurring in patients with PMGCT are thought to be primary, and not therapy related. [11][12][13][14] In some cases, immunohistochemical and cytogenetic evidence (especially presence of i12p chromosome) had previously suggested the clonal relationship between the PMGCT and the hematologic malignancy. [11][12][13][14] As in other previously reported cases, the short interval between the PMGCT and the occurrence of the hematopoietic malignancy argues against a therapy-related origin of the acute megakaryocytic leukemia.…”

mentioning

confidence: 99%

“…[11][12][13][14] In some cases, immunohistochemical and cytogenetic evidence (especially presence of i12p chromosome) had previously suggested the clonal relationship between the PMGCT and the hematologic malignancy. [11][12][13][14] As in other previously reported cases, the short interval between the PMGCT and the occurrence of the hematopoietic malignancy argues against a therapy-related origin of the acute megakaryocytic leukemia. There have been two reported cases of PMGCT complicated by acute megakaryotic leukemia, one presenting with an extradural mass and the other with organomegaly.…”

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confidence: 99%

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Metastasis of a Histologically Benign–Appearing Meningioma to the Iliac Bone

Azene

Gai

Tabar

et al. 2008

JCO

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lies in the presence of hematologic precursors within the PMGCT stroma and vessels within the yolk sac tumor component of these tumors. 11 It can be further speculated that expression of hematopoietic growth and differentiation factors in some PMGCT could drive the differentiation of primordial germ cells into hematopoietic progeny. The differentiation factors involved may be responsible for the preferred commitment of the transformed precursors to megakaryocytic and monocytic lineage. Additionally, in a few cases of PMGCT-associated leukemia in which the immunophenotype of the leukemic cells was compared with that of the intratumoral hematopoietic precursors, a comparable result was observed. This suggests a spread of hematopoietic tumor cells from the PMGCT to blood, bone marrow, and/or extramedullary sites. 12 The median time for the development of hematologic neoplasia associated with PMGCT is 6 months (range, 0 to 47 months). 3 The clinical course of the hematologic neoplasia is very aggressive with a median survival of 5 months (range, 0 to 16 months) after diagnosis. 3 Hematologic malignancies occurring in patients with PMGCT are thought to be primary, and not therapy related. [11][12][13][14] In some cases, immunohistochemical and cytogenetic evidence (especially presence of i12p chromosome) had previously suggested the clonal relationship between the PMGCT and the hematologic malignancy. [11][12][13][14] As in other previously reported cases, the short interval between the PMGCT and the occurrence of the hematopoietic malignancy argues against a therapy-related origin of the acute megakaryocytic leukemia. There have been two reported cases of PMGCT complicated by acute megakaryotic leukemia, one presenting with an extradural mass and the other with organomegaly. 8 However, our patient is the first, to our knowledge, to be ever reported in English literature of a PMGCT evolving into an extramedullary acute megakaryocytic leukemia manifesting as a mass causing cord compression. Furthermore, the aggressive course of the acute megakaryoblastic leukemia involving the CNS with sparing of the bone marrow is striking.

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Section: Metastasis Of a Histologically Benign-appearing Meningioma T...mentioning

confidence: 99%

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confidence: 99%

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confidence: 99%

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confidence: 99%

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Metastasis of a Histologically Benign–Appearing Meningioma to the Iliac Bone

Azene

Gai

Tabar

et al. 2008

JCO

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“…Contrasting to meningiomas of the axial skeleton, intraosseous meningiomas of the appendicular skeleton are reported with much less frequency. To our knowledge, there are only a sparse number of reports of intraosseous meningiomas within the appendicular skeleton [6][7][8]. Additionally, intraosseous meningiomas generally present with pain and swelling leading to a wide differential diagnosis, including both primary and secondary bone cancers, which warrant further clinical investigation [6,8,9].…”

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confidence: 99%

Asymptomatic Intraosseous Meningioma of the Humerus: A Case Report and Review of the Literature

Patel¹,

McKnight²,

Vyas³

2022

Cureus

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Meningiomas are the most common central nervous system tumor. They are typically benign neoplasms but may produce neurological symptoms due to mass effect. Meningiomas may also extend to extradural locations; however, these account for only a small percentage of all meningiomas. Most extradural meningiomas arise in intraosseous locations, usually within the cranial bones or vertebrae. However, this is a rare case of extradural extension of an asymptomatic intracranial meningioma to the proximal humerus in the absence of any musculoskeletal symptoms. To the best of our knowledge, this presentation of an extradural intraosseous meningioma has not previously been reported in the literature. We present a case of an incidental intraosseous meningioma in a 66-year-old man. This patient was initially being screened for metastasis of stage IA1 adenocarcinoma of the lung, and a positron emission tomography (PET) scan revealed a focus of activity in the proximal diaphysis of the right humerus suspicious for malignancy. The upper extremity magnetic resonance imaging (MRI) demonstrated an indeterminate lesion. Curettage of the humeral lesion revealed an intraosseous psammomatous meningioma without evidence of metastatic lung carcinoma. Our case report aims to illustrate the importance of considering alternative metastatic sources, such as intracranial meningioma, during the investigation of an indeterminate bony lesion. This is the first case to illustrate asymptomatic intraosseous meningioma in an appendicular skeletal location, highlighting the need for thorough source investigation.

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Treatment recommendations for primary extradural meningiomas

Mattox

Hughes

Oleson

et al. 2010

Cancer

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Highly reflective, surface‐metalized, flexible polyimide films were prepared by the incorporation of a soluble silver‐ion complex, (hexafluoroacetylacetonato)silver(I) (AgHFA), into dimethylacetamide solutions of poly(amic acid) prepared from 2,2‐bis(3,4‐dicarboxyphenyl)hexafluoropropane dianhydride and 2,2‐bis[4‐(4‐aminophenoxy)phenyl]hexafluoropropane. The thermal curing of solution‐cast silver(I)–poly(amic acid) films to 300°C led to cycloimidization of the amic acid with concomitant silver(I) reduction and the formation of a reflective, air‐side‐silvered surface at very low (2 wt % and 0.3 vol %) silver concentrations. The reflective surface evolved only when the cure temperature reached about 275°C, although X‐ray diffraction showed metallic silver in the hybrid film by 200°C. After a maximum specular reflectivity greater than 80% was achieved for the 2 wt % silver film, the specular reflectivity diminished sharply with further heating at a constant temperature of 300°C. Incorporating the AgHFA complex into the soluble, fully imidized form of poly{(1,3‐dihydro‐1,3‐dioxo‐2H‐isoindole‐2,5‐diyl)[2,2,2‐trifluoro‐1‐(trifluoromethyl)ethylidene](1,3‐dihydro‐1,3‐dioxo‐2H‐isoindole‐5,2‐diyl)‐1,4‐phenyleneoxy‐1,4‐phenylene[2,2,2‐trifluoro‐1‐(trifluoromethyl)ethylidene]‐1,4‐phenyleneoxy‐1,4‐phenylene} gave films that were 25% less reflective than those beginning with poly(amic acid). Though highly reflective, the films were not electrically conductive. The metalized membranes were thermally stable and maintained mechanical properties similar to those of the parent polyimide. Transmission electron microscopy revealed an air‐side, near‐surface layer of silver that was about 40 nm thick; the interior of the film had well‐dispersed metal particles with diameters mostly less than 2 nm. The near‐surface silver layer maintained its integrity because of physical entrapment of the metal nanoparticles beneath a thin layer of polyimide; that is, the practical adhesion of the metal layer was good. © 2006 Wiley Periodicals, Inc. J Appl Polym Sci 103: 2409–2418, 2007

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Meningioma of the scapula

Cited by 8 publications

References 7 publications

Metastasis of a Histologically Benign–Appearing Meningioma to the Iliac Bone

Metastasis of a Histologically Benign–Appearing Meningioma to the Iliac Bone

Asymptomatic Intraosseous Meningioma of the Humerus: A Case Report and Review of the Literature

Treatment recommendations for primary extradural meningiomas

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