Maternal Hypothyroidism-Milk Ejections: What is the Link?

Maternal thyroid hormones (THs; 3,5,3'-triiodothyronine (T3) and thyroxin (T4)) exert vital roles throughout the development and show a permissive role in the neurological functions, in particular the sensorineural actions. The actions of THs can be mediated through the genomic and non-genomic pathways. On the other hand, there are associations between the maternal hypothyroidism and neonatal sensorineural deficits. A gestational hypothyroidism displays sensory and motor deficits, grave cognitive, permanent long-lasting variations in child brain structure, and neurodevelopmental disorders. During prenatal and early postnatal periods, any disruption in the levels of T4 or T3 can lead to irreversible disorders in sensorimotor and intelligence function, psychotic behavior, mental retardation, depression, coma and several morphological variations in the brain.

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Maternal Hyperthyroidism - Milk Ejections: What is the Association?

Rg¹

2018

TTSR

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Maternal Hypothyroidism-Milk Ejections: What is the Link?

Abstract: HYPOTHESISThe normal activity for the maternal hypothalamic-pituitary-thyroid axis (HPTA) isdynamic for the developing fetuses and neonates(Ahmed,

Cited by 5 publications

References 2 publications

Maternal Thyroid Dysfunction, Intrauterine and Fetal Growth Restriction

Maternal Thyroid Dysfunction, Intrauterine and Fetal Growth Restriction

Maternal Hypothyroidism and Sensorineural Disability

Maternal Hyperthyroidism - Milk Ejections: What is the Association?

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