Massive acquired cholesteatoma of the temporal bone

Wright, J. L. W.; Colman, B. H.; Connor, A. F.

doi:10.1017/s0022215100082037

Cited by 6 publications

(3 citation statements)

References 10 publications

(3 reference statements)

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Previous histological reports on temporal bones of patients who have died from leukaemia (Schukneckt, 1974) have shown variable invasion of the mastoid air cell system by leukaemic tissue, assoiated with focal haemorrhages in the middle ear mucosa and in the membranous structures of the inner ear. Massive bone erosion with total destruction of the labyrinth may occur at a late stage (Wright et al, 1976). Computerized tomographic views of the temporal bone showed bone destruction and invasion of the posterior cranial fossa by leukaemic tissue in the present case (Fig.…”

Section: Discussionsupporting

confidence: 49%

Mastoid chloroma as relapse in acute myeloid leukaemia

Chapman¹,

Johnson²

1980

J. Laryngol. Otol.

View full text Add to dashboard Cite

show abstract

Section: Discussionsupporting

confidence: 49%

Mastoid chloroma as relapse in acute myeloid leukaemia

Chapman¹,

Johnson²

1980

J. Laryngol. Otol.

View full text Add to dashboard Cite

show abstract

“…The criterion for inclusion in the study was surgically documented evidence of acquired aural cholesteatoma extending into the middle and/or posterior cranial fossae. The criteria of Derlacki and Clemis 16 for congenital cholesteatoma were used to make the distinction between primary and acquired cholesteatoma. These authors noted the following points in defining congenital cholesteatoma of the tympanum and mastoid: 1) development of the cholesteatoma behind an intact tympanic membrane, 2) absence of previous history of aural infections, and 3) no demonstrable connection of the cholesteatoma with the external auditory canal.…”

Section: Methodsmentioning

confidence: 99%

“…In 1981 Yanagihara and Matsumoto 15 presented six patients with extensive cholesteatoma in the petrous apex and argued that only one of these patients possibly met the criteria defined by Derlacki and Clemis 16 for primary cholesteatoma of the temporal bone. After reviewing the series of both Tytus and Pennybacker and Cawthorne and Griffith, the researchers further argued that several patients in those series did not meet the Derlacki and Clemis criteria for primary cholesteatoma.…”

Section: Introductionmentioning

confidence: 99%

Intracranial Extension of Acquired Aural Cholesteatoma

Horn

2000

The Laryngoscope

View full text Add to dashboard Cite

Objective: Cholesteatoma of the petrous bone extending into the intracranial region is an unusual occurrence. Most cases have been attributed to secondary extension of a primary epidermal blastomatous malformation of the temporal bone into the middle or posterior fossae. Within the past two and a half decades, intracranial extension of acquired aural cholesteatoma has been recognized as a likely alternative to this mechanism. Recent literature has rejoined this observation by considering both primary and secondary cholesteatoma of the petrous bone as a single group, petrosal cholesteatoma. The present study is presented to analyze the clinical presentation, imaging findings, and surgical treatment of six patients with acquired aural cholesteatoma extending into the intracranial region. Findings in this study are compared with the extant literature on congenital and acquired cholesteatoma of the petrous bone. This study proposes that petrosal cholesteatoma is a valid anatomical construct; however, the pathogenesis of petrosal cholesteatoma is still important in understanding the clinical presentation and management of cholesteatoma that extends beyond the usual confines of the middle ear and mastoid. Study Design: Retrospective case review conducted at a tertiary referral center. Methods: From 1985 to 1999, 477 patients were surgically treated for acquired aural cholesteatoma. Patients with intracranial extension of cholesteatoma were studied. Clinical presentation, imaging studies, operative findings, surgical treatment, and postoperative results were evaluated. Results: Six cases in a series of 477 patients with acquired aural cholesteatoma had intracranial extension of disease. In this series, the most frequent pathway for intracranial extension was supralabyrinthine through the supratubal recess into the middle cranial fossa. A less frequent pathway was via the retrofacial air cells into the posterior cranial fossa. Surgical access for removal of intracranial cholesteatoma was accomplished through several approaches including translabyrinthine, transcochlear, retrolabyrinthine, and middle cranial fossa. In two patients who had reoperation for possible residual disease, one was free of residual disease and one was found to have residual cholesteatoma in the region of the horizontal facial nerve. Conclusion: Acquired aural cholesteatoma can extend into either the middle or posterior cranial fossae. In this study, cholesteatoma extended into the middle fossa through the supratubal recess along the labyrinthine facial nerve and into or above the internal auditory canal. A less frequent path is through the retrofacial air cells into the posterior fossa. Intracranial acquired cholesteatoma is generally small and presents with complaints related to underlying otitis media rather than the neurological deficits that are often associated with primary petrous bone cholesteatoma. While computed tomography and magnetic resonance imaging are both required to differentiate congenital petrous cholesteatoma from other lesions...

show abstract

Otogenic intracranial hypertension

O’Connor

Moffat

1978

J. Laryngol. Otol.

View full text Add to dashboard Cite

Massive acquired cholesteatoma of the temporal bone

Cited by 6 publications

References 10 publications

Mastoid chloroma as relapse in acute myeloid leukaemia

Mastoid chloroma as relapse in acute myeloid leukaemia

Intracranial Extension of Acquired Aural Cholesteatoma

Otogenic intracranial hypertension

Contact Info

Product

Resources

About