Manganese Pneumonitis

DAVIES, T A L

doi:10.1136/oem.3.3.111

Cited by 6 publications

(6 citation statements)

References 2 publications

(2 reference statements)

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“…There was more radiographic evidence of disease due to exposure to asbestos (Kilburn and Warshaw, 1990), to hard-metal grinding tools (cobalt) (Sprince et al, 1988), or to manganese (steel welding, Davies, 1946) in workers at Tinker AFB than in the unexposed group, in keeping with their workplace exposures. Jet engine repair workers had impaired balance, simple and choice reaction time, blink reflex latency, and color discrimination and excess frequencies of neurobehavioral symptoms.…”

Section: Discussionmentioning

confidence: 81%

Neurobehavioral and Respiratory Findings in Jet Engine Repair Workers: A Comparison of Exposed and Unexposed Volunteers

Kilburn

1999

Environmental Research

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Section: Discussionmentioning

confidence: 81%

Neurobehavioral and Respiratory Findings in Jet Engine Repair Workers: A Comparison of Exposed and Unexposed Volunteers

Kilburn

1999

Environmental Research

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“…In 1946, a British researcher reported a clear-cut cluster of pneumonia in a factory producing potassium permanganate. 53 The clinical picture was that of a febrile lobar pneumonia, but also of severe respiratory irritation with bronchitis, pharyngitis, and epistaxis. The outbreak occurred in a manufacturing facility where manganese dioxide was ground, reacted with potassium hydroxide and lime, roasted in rotary kilns, and then purified through electrolysis.…”

Section: Pneumonia or Chemical Pneumonitis?mentioning

confidence: 99%

Pneumococcal pneumonia on the job: Uncovering the past story of occupational exposure to metal fumes and dust

Naidoo

Blanc

2022

American J Industrial Med

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The objectives of this study are to elucidate the early history of risk for pneumococcal pneumonia from occupational exposure to metal fumes and dusts, and to demonstrate the importance of searching older literature when performing reviews. We performed manual searching for articles in the Library of the Surgeon General's Office (the precursor to Index Medicus), in the Hathi Trust database, in PubMed, andby screening reference lists in literature appearing before the introduction of PubMed. An early body of literature, from the 1890s onward, recognized that pneumonia was linked to "Thomas slag," a steel industry byproduct containing iron, manganese, and lime. Researchers, mainly in Germany, showed that workers in metal-dust-exposed occupations, especially using manganese, manifested an increased incidence of pneumococcal pneumonia. An outbreak of pneumococcal pneumonia in the 1930s implicated manganese fume in its etiology. In the immediate post-World War II period, there was a brief flurry of interest in pneumonia from exposure to potassium permanganate that was soon dismissed as a chemical pneumonitis. After a hiatus of two decades, epidemiologic investigations drew attention to the pneumonia risks of welding and related metal fume exposure, bringing renewed interest to the forgotten role of pneumococcal pneumonia as an occupational disease. Occupational or environmental inhalation of manganese, iron, or irritants may be causally related to increased pneumococcal pneumonia risk. In particular, the risk associated with manganese seems to be overlooked in recent literature. An important conclusion is the importance of obtaining additional evidence through a deeper assessment of the literature in a broad historical context.

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“…However, pulmonary disturbances may continue; a manganese pneumonitis has been described [154,155]. Manganese is used to alloy steels, as a coloring agent in glass and ceramic manufacture, and as a flux component in welding operations.…”

Section: Toxic Gas Inhalationmentioning

confidence: 99%

Respiratory Effects of Short-Term, High-Intensity Toxic Inhalations: Smoke, Gases, and Fumes

Wald

Balmes

1987

J Intensive Care Med

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Respiratory effects of short-term, high-intensity toxic inhalations: smoke, gases, and fumes. J Intensive Care Med 1987;2:260-278.Respiratory insufficiency resulting from inhalation of hot air, smoke, or toxic gas is a common cause of death in fire victims. Toxic gas inhalation in settings other than fires is a less common but still important cause of death and disability. Pulmonary inhalation injury and systemic intoxication from exposure to a variety of agents are discussed with regard to their pathogenesis, pathophysiology, diagnosis, and treatment. Upper airway obstruction and noncardiogenic pulmonary edema are the lifethreatening respiratory complications in the immediate postinhalation period. Carbon monoxide and hydrogen cyanide intoxication are frequently associated with inhalation injury in fire victims. Early recognition and treatment of inhalation injury and systemic intoxication in victims of fires and industrial and environmental accidents could well result in improved survival.

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Manganese Pneumonitis

Cited by 6 publications

References 2 publications

Neurobehavioral and Respiratory Findings in Jet Engine Repair Workers: A Comparison of Exposed and Unexposed Volunteers

Neurobehavioral and Respiratory Findings in Jet Engine Repair Workers: A Comparison of Exposed and Unexposed Volunteers

Pneumococcal pneumonia on the job: Uncovering the past story of occupational exposure to metal fumes and dust

Respiratory Effects of Short-Term, High-Intensity Toxic Inhalations: Smoke, Gases, and Fumes

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