Macular Edema in Chronic Vogt-Koyanagi-Harada Syndrome

Risk factors for poor visual outcome following cataract surgery in Vogt-Koyanagi-Harada disease

“…In this study, the incidence of CMO in the uveitic eyes of 12% at 1 month and 8% at 3 months15 was similar to our CMO rates. This suggests that the CMO in our patients may be a result of cataract surgery rather than from VKH disease and is in keeping with the fact that CMO is an uncommon complication of VKH disease,16 especially if surgery is performed after a period of quiescence of 3 months in combination with the judicious use of prophylactic corticosteroids. Although the incidence of CMO is low, it is nonetheless an important cause of poor BCVA postsurgery (2/7, 28.6%), and prophylactic corticosteroid treatment does not entirely prevent its occurrence.…”

Vogt–Koyanagi–Harada disease in Hispanic patients

“…There are many possible causes of decreased vision in patients with VKH. During episodes of initial or recurrent uveitis, vision may be limited by the inflammation itself, by serous elevation of the neurosensory retina, or, rarely, by cystoid macular edema [15]. Once the inflammation has been controlled, complications such as subretinal fibrosis may develop as long-term complications of VKH [16].…”

Vogt–Koyanagi–Harada syndrome – current perspectives

“…Subretinal fibrosis can occur in up to 40% of VKHD patients165 and may lead to substantial visual impairment when the fovea is involved 166. Unlike other ocular inflammatory conditions, CME appears to be a relatively rare complication of VKHD 160,167. Other uncommon complications (Table 1) includes posterior synechiae,158 rhegmatogenous retinal detachment and ERM160 and MH formation,84 band keratopathy,168 and pseudotumoral RPE proliferation 169,170.…”