“…Iglauer and Molt [3] first described the nasogastric tube syndrome in 1939 in a series of 12 patients. Holinger and Loeb [4] described a further 4 cases in 1946.…”
Nasogastric tube syndrome is an uncommon but a potentially life-threatening complication of nasogastric intubation. It presents as an acute upper airway obstruction secondary to bilateral abductor vocal cord dysfunction resulting from postcricoid chondritis. However, a unilateral variant has also been reported and may herald the development of bilateral pathology. The purpose of this report is to highlight early warning signals of impending crisis. Clinical Presentation: A case of unilateral vocal cord involvement secondary to postcricoid chondritis resulting from nasogastric intubation is presented. An additional feature, not reported previously, is the presence of two inflammatory nodular swellings over the cricoid cartilage.
“…Iglauer and Molt [3] first described the nasogastric tube syndrome in 1939 in a series of 12 patients. Holinger and Loeb [4] described a further 4 cases in 1946.…”
Nasogastric tube syndrome is an uncommon but a potentially life-threatening complication of nasogastric intubation. It presents as an acute upper airway obstruction secondary to bilateral abductor vocal cord dysfunction resulting from postcricoid chondritis. However, a unilateral variant has also been reported and may herald the development of bilateral pathology. The purpose of this report is to highlight early warning signals of impending crisis. Clinical Presentation: A case of unilateral vocal cord involvement secondary to postcricoid chondritis resulting from nasogastric intubation is presented. An additional feature, not reported previously, is the presence of two inflammatory nodular swellings over the cricoid cartilage.
“…Despite extensive adaptation, serious complications have occasionally been reported. Iglauer and Molt first described damage to the larynx due to an indwelling enteric tube in a case series in 1939 [2] . NTS was later formally defined by Sofferman et al [3] in 1990.…”
“…Beschreibungen dieses Mechanismus gehen bis in das Jahr 1939 zurück [34]. Iglauer [1,3,4,14,18,21,24,26,32,40,51,58,67,76,79,80] ryngoskopisch gesicherten Stimmlippenstillstand angegeben [38].…”
Since the phoniatrician H. Bauer described the first case of recurrent laryngeal nerve palsy most likely caused by intubation some 45 years ago, several case reports have been published. However, systematic analyses regarding the frequency of recurrent laryngeal nerve palsies due to intubation are scarce, and none of them has used the proper methods to demonstrate clearly that such a mechanism exists. Currently available data justify the assumption that not every recurrent laryngeal nerve palsy following thyroid surgery is due to the operation itself and that the damage caused by intubation, however, may only account for a minority of these cases. The differential diagnosis of postoperative recurrent laryngeal nerve palsy requires the use of specific tools which go beyond simple laryngoscopy and include stroboscopy as well as intra- and extralaryngeal electromyography. A partial palsy of recurrent laryngeal nerve due to intubation would be associated with severe dysphonia or aphonia, not with dyspnea because of the typical intermediate position of the paralyzed vocal folds with a normal electromyographic function of the cricothyroid muscle. The use of these methods to identify the nature of postoperative recurrent laryngeal nerve palsy is recommended in cases of regular intraoperative neuromonitoring but postoperatively impaired function of the vocal cords.
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