LVI Bilateral Jugular Ligation following Bilateral Suppurative Mastoiditis

Evans, Maurice G.

doi:10.1177/000348944205100306

Cited by 15 publications

(4 citation statements)

References 5 publications

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“…Four other aspects of the possible link between PTS and cranial venous outflow obstruction were also documented by this time. First, Evans [42] gave further evidence of PTS following internal jugular vein ligation although in reviewing other reports as well as his own cases he found that none of the 7 patients having bilateral ligation for non-otitic problems developed papilloedema whereas the 3 cases who had the procedure in relation to ear disease did. Of 6 cases who had unilateral ligation for bilateral ear disease only one developed papilloedema.…”

Section: Historical Perspectivementioning

confidence: 99%

Cranial Venous Outflow Obstruction and Pseudotumor Cerebri Syndrome

Owler

Parker

Hálmagyi

et al. 2005

Advances and Technical Standards in Neurosurgery

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Section: Historical Perspectivementioning

confidence: 99%

Cranial Venous Outflow Obstruction and Pseudotumor Cerebri Syndrome

Owler

Parker

Hálmagyi

et al. 2005

Advances and Technical Standards in Neurosurgery

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“…Two other important developments were the introduction of direct sinography 1937 27 and the recognition that extracranial (internal jugular) venous obstruction could cause PTC, both with ligation in the treatment of ear infection or in other conditions, as first documented by Evans. 23 Attention to this aspect was, however, somewhat sporadic over the following decades, although there were two important papers, in the first of which 69 the authors focused on diagnosis and treatment and in the second of which 42 the authors focused particularly on mechanism.…”

Section: Cranial Venous Outflow Impairment-the Next Period?mentioning

confidence: 99%

The historical development of the pseudotumor concept

Johnston¹

2001

FOC

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The author outlines the history of the development of the pseudotumor concept, first specifically formulated by Nonne in 1904 but implicit in earlier descriptions. A scheme of periodization is applied, identifying otological, neurosurgical, neurological, and neuroophthalmological periods; he relates theoretical and practical developments in the understanding of the condition to the concerns of the dominant speciality involved in diagnosis and management. Key studies from each period are discussed in relation to the issues of mechanism, origin, nomenclature, diagnosis, and treatment. During the otological period, approaches to the condition were heavily influenced by its common association with middle ear infection, often with venous sinus involvement, and this led to the idea of disturbed cerebrospinal fluid dynamics as the cause. The advent of neuroradiological systems ushered in the neurosurgical period in which the significant but unfinished debate began: which intracranial compartment is primarily implicated in the intracranial hypertension? By the time of the neurological period cerebral edema was implicated as the cause, and this coincided with the availability of appropriate treatment methods and steroidal and diuretic agents. Toward the end of this period additional major advances in investigative methods again brought the issue of mechanism into contention, although a resolution was not to follow. The neuroophthalmological period is characterized by an increased awareness of the significance of visual loss and a return to one of the original methods of treatment. Finally, the possible significance of impaired cranial venous outflow, a thread running through all periods, is considered.

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“…6 However, if bilateral internal jugular vein resection is required due to tumor invasion, the consequent disruption of venous return and increased intracranial pressure can lead to fatal complications. 7,8 Complications of perfusion failure in the IJV, including blindness, syndrome of inappropriate antidiuretic hormone secretion airway obstruction, cerebral infarction and even death have been reported. 1,2 To avoid these complications, a two-stage neck dissection or simultaneous reconstruction of the IJV is recommended.…”

Section: Discussionmentioning

confidence: 99%

Reconstruction for Bilateral Internal Jugular Vein Perfusion Disruption

et al. 2023

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When resecting the internal jugular veins bilaterally in surgery for head and neck cancer, it is necessary to perform neck dissection in two stages or to reconstruct the internal jugular veins in one stage. Reconstruction of the internal jugular vein using grafting or direct anastomosis to the external jugular vein have both been reported. We report the case of a 53-year-old man with accidental injury to the left internal jugular vein after resection of the right internal jugular vein for supraglottic cancer. The left internal jugular vein was damaged near the inflow of the subclavian vein, making vein grafting difficult. Therefore, internal jugular venous return was reestablished by end-to-side anastomosis of the left internal jugular vein to the left external jugular vein system. In this surgical procedure, by incising the internal jugular vein obliquely, it was not necessary to match the calibers of the internal jugular vein and the external jugular vein system, and a smooth hemodynamic body was reconstructed. In addition, we were able to reconstruct the internal jugular vein while preserving blood flow in the external jugular vein system. End-to-side anastomosis of the internal jugular vein to the external jugular system is an option for internal jugular vein reconstruction.

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LVI Bilateral Jugular Ligation following Bilateral Suppurative Mastoiditis

Cited by 15 publications

References 5 publications

Cranial Venous Outflow Obstruction and Pseudotumor Cerebri Syndrome

Cranial Venous Outflow Obstruction and Pseudotumor Cerebri Syndrome

The historical development of the pseudotumor concept

Reconstruction for Bilateral Internal Jugular Vein Perfusion Disruption

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