2016
DOI: 10.1038/s41598-016-0029-9
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Lower and upper bounds for entanglement of Rényi-α entropy

Abstract: Entanglement Rényi-α entropy is an entanglement measure. It reduces to the standard entanglement of formation when α tends to 1. We derive analytical lower and upper bounds for the entanglement Rényi-α entropy of arbitrary dimensional bipartite quantum systems. We also demonstrate the application our bound for some concrete examples. Moreover, we establish the relation between entanglement Rényi-α entropy and some other entanglement measures.

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Cited by 10 publications
(7 citation statements)
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“…In the future, identification of underlying mechanisms by which cancer cells interact with ECs to promote extravasation will definitely lead to the development of new therapies to reduce metastasis. Intriguingly, recent data indicated that tumor cells implanted into the brain of nude mice spread along the abluminal surface of blood vessels instead of the bloodstream [ 182 ], which raises the possibility and likelihood of an alternative mechanism of dissemination. If other pathways besides the blood and lymphatic vessels for tumor dissemination are validated, then current therapeutics targeting cancer cells in circulation may be less effective.…”
Section: Discussionmentioning
confidence: 99%
“…In the future, identification of underlying mechanisms by which cancer cells interact with ECs to promote extravasation will definitely lead to the development of new therapies to reduce metastasis. Intriguingly, recent data indicated that tumor cells implanted into the brain of nude mice spread along the abluminal surface of blood vessels instead of the bloodstream [ 182 ], which raises the possibility and likelihood of an alternative mechanism of dissemination. If other pathways besides the blood and lymphatic vessels for tumor dissemination are validated, then current therapeutics targeting cancer cells in circulation may be less effective.…”
Section: Discussionmentioning
confidence: 99%
“…Data from this study suggest that antigen-specific pTregs (but not tTregs) are liable to dysfunction in an inflammatory microenvironment, such as occurs in a high-risk host bed. This distinction between tTregs and pTregs is of critical importance, as it supports the proposition that pTregs maintain peripheral tolerance to allografts in the low-risk setting, but their function is subverted by graft site inflammation, in which case they promote rejection [118]. …”
Section: Tregs In Corneal Transplantationmentioning
confidence: 63%
“…Indeed, in an investigation of the function of tTregs and pTregs in a high-risk model of corneal transplantation, it has been demonstrated that the frequency and function of pTregs were suppressed in high-risk transplants and that this corresponded to reduced expression of IL-10 and TGF-β [118]. Data from this study suggest that antigen-specific pTregs (but not tTregs) are liable to dysfunction in an inflammatory microenvironment, such as occurs in a high-risk host bed.…”
Section: Tregs In Corneal Transplantationmentioning
confidence: 99%
“…A few previous attempts for entanglement upper bounds were limited to specific entanglement measures (for example, the entanglement of formation in [14] and the Rényi-α entropy in [15]). These usually cannot be extended to more complicated systems, multipartite entanglement being a key example.…”
mentioning
confidence: 99%