Low serum potassium level is associated with nonalcoholic fatty liver disease and its related metabolic disorders

Abstract

Low serum potassium level significantly associated with prevalence of NAFLD in middle-aged and elderly Chinese.

Hypertension and non‐alcoholic fatty liver disease proven by transient elastography

“…A total of 39.83% of the subjects had been diagnosed with NAFLD. Our findings are inconsistent with another study that reported a lower prevalence of NAFLD (19–34) . A possible explanation for the different prevalence may originate from the characteristics of the studied populations.…”

“…Our findings are inconsistent with another study that reported a lower prevalence of NAFLD (19)(20)(21)(22)(23)(24)(25)(26)(27)(28)(29)(30)(31)(32)(33)(34). 11,15,16 A possible explanation for the different prevalence may originate from the characteristics of the studied populations. Most of the subjects in our study (from Hunan province, China) usually lived on a spicy, high salt diet, which may contribute to the results.…”

Quantifying the dietary potassium requirement of juvenile grass carp (Ctenopharyngodon idellus)

“…The direct relation between dietary potassium and liver function is still unclear. However, blood potassium level may be used as an indicator of impending liver problems and low blood potassium level can increase the risk of having nonalcoholic fatty liver disease (Sun et al, 2014). Whether higher dietary potassium would cause liver dysfunction of fish needs further investigation.…”

Effect of interleukin‐2 receptor antagonists on new‐onset diabetes after liver transplantation: A retrospective cohort study

“…Previous findings indicated that age, hepatitis C virus infection, BMI, family history of diabetes, immunosuppressant regimen, preoperative FPG level, acute rejection, preoperative liver function, severity of liver cirrhosis, cytomegalovirus infection, and metabolic syndrome, including hypertension and hyperlipidemia, were risk factors for NODAT. [23][24][25][26][27][28][29][30][31][32][33][34][35][36][37][38] The findings from the present study indicate that sex, hepatitis virus infection, donor type, BMI, history of hypertension, preoperative total cholesterol and triglyceride levels, and severity of liver cirrhosis were similar between the IL-2Ra and non-IL-2Ra groups. In contrast, age, preoperative liver function, preoperative FPG level, initial immunosuppressant regimen type, and CsA levels after liver transplantation differed between these two groups and may have affected the estimation of the effect of IL-2Ra on NODAT.…”