Long face: New proposals for taxonomy, diagnosis, treatment

Merville, L; Diner, P.A.

doi:10.1016/s1010-5182(87)80024-0

Cited by 9 publications

(13 citation statements)

References 6 publications

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“…Interestingly, in this study the patients required surgical correction of the Class III condition, and although all subjects had a degree of mandibular prognathism, only 14% exhibited maxillary retrognathism. To be more exact in diagnosis and treatment planning, Merville and Diner (1987) suggested new proposals for taxonomy of facial types based upon the underlying perturbation such as hyposoprosopia (''long face''). Nevertheless, other terminology such as hyperdivergent and normodivergent facial patterns appears in the orthodontic, surgical, and dental literature (e.g., Joseph et al, 1998) and there appears to be little universal uniformity in this regard.…”

Section: Heterogeneitymentioning

confidence: 99%

Morphologic determinants in the etiology of class III malocclusions: A review

Singh

1999

Clin. Anat.

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Morphospatial disharmony of the craniomaxillary and mandibular complexes may yield apparent mandibular prognathism, but Class III malocclusions can exist with any number of aberrations of the craniofacial complex. Deficient orthocephalization of the cranial base allied with a smaller anterior cranial base component has been implicated in the etiology of Class III malocclusions. Whereas the more acute cranial base angle may affect the articulation of the condyles resulting in their forward displacement, the reduction in anterior cranial size may affect the position of the maxilla. As well, intrinsic skeletal elements of the maxillary complex may be responsible for maxillary hypoplasia that may exacerbate the anterior crossbite seen in the Class III condition. Conversely, with an orthognathic maxilla, condylar hyperplasia and anterior positioning of the condyles at the temporo-mandibular joint may produce an anterior crossbite. Aside from the skeletal components, soft tissue matrices, particularly labial pressure from the circumoral musculature, may influence the final outcome of craniofacial growth of a child skeletally predisposed to Class III conditions. Indeed, as some Asian ethnic groups demonstrate an increased prevalence of Class III malocclusions, it is likely that the skeletal components and soft tissues matrices are genetically determined. Presumably, the co-morphologies of the craniomaxillary and mandibular complexes are likely dependent upon candidate genes that undergo gene-environmental interactions to yield Class III malocclusions. The identification of such genes is a desirable step in unraveling the complexity of Class III malocclusions. With this knowledge, the clinician may elect an early course of dentofacial orthopedic and orthodontic treatments aimed at preventing the development of Class III malocclusions.

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Section: Heterogeneitymentioning

confidence: 99%

Morphologic determinants in the etiology of class III malocclusions: A review

Singh

1999

Clin. Anat.

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“…Mackay et al (1992) studied craniofacial form in Class III cases and identified five subgroups. To be more exact in diagnosis and treatment planning, Merville and Diner (1987) suggested new proposals for taxonomy of facial types based upon the underlying perturbation such as hyposoprosopia (''long face''). To be more exact in diagnosis and treatment planning, Merville and Diner (1987) suggested new proposals for taxonomy of facial types based upon the underlying perturbation such as hyposoprosopia (''long face'').…”

Section: Heterogeneitymentioning

confidence: 99%

Morphologic determinants in the etiology of class III malocclusions: A review

Singh

1999

Clin. Anat.

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Morphospatial disharmony of the craniomaxillary and mandibular complexes may yield apparent mandibular prognathism, but Class III malocclusions can exist with any number of aberrations of the craniofacial complex. Deficient orthocephalization of the cranial base allied with a smaller anterior cranial base component has been implicated in the etiology of Class III malocclusions. Whereas the more acute cranial base angle may affect the articulation of the condyles resulting in their forward displacement, the reduction in anterior cranial size may affect the position of the maxilla. As well, intrinsic skeletal elements of the maxillary complex may be responsible for maxillary hypoplasia that may exacerbate the anterior crossbite seen in the Class III condition. Conversely, with an orthognathic maxilla, condylar hyperplasia and anterior positioning of the condyles at the temporo‐mandibular joint may produce an anterior crossbite. Aside from the skeletal components, soft tissue matrices, particularly labial pressure from the circumoral musculature, may influence the final outcome of craniofacial growth of a child skeletally predisposed to Class III conditions. Indeed, as some Asian ethnic groups demonstrate an increased prevalence of Class III malocclusions, it is likely that the skeletal components and soft tissues matrices are genetically determined. Presumably, the co‐morphologies of the craniomaxillary and mandibular complexes are likely dependent upon candidate genes that undergo gene‐environmental interactions to yield Class III malocclusions. The identification of such genes is a desirable step in unraveling the complexity of Class III malocclusions. With this knowledge, the clinician may elect an early course of dentofacial orthopedic and orthodontic treatments aimed at preventing the development of Class III malocclusions. Clin. Anat. 12:382–405, 1999. © 1999 Wiley‐Liss, Inc.

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“…Ä hnliche kraniofaziale Folgen wurden auch bei einer Hypertrophie der Tonsillen und bei Patienten mit dem sogenannten " long face syndrome " gefunden [16] . Dieser Terminus beschreibt die exzessiv vergr ö ß erte vertikale Dimension des Gesichtes, die entweder durch einen stark vergr ö ß erten Kieferwinkel oder einen poste rioren maxill ä ren dento-alveol ä ren Exzess verursacht wird [17,18] ( • ▶ Abb. 1a -b ).…”

Section: Interaktion Von Hals-nasen-ohren-heilkunde Und Kieferorthop unclassified

Interaktion von Hals-Nasen-Ohren-Heilkunde und Kieferorthopädie: Die Beziehung zwischen dem nasopharyngealen Luftraum und den kraniofazialen Strukturen

Stellzig‐Eisenhauer¹,

Meyer-Marcotty²

2010

Laryngo-Rhino-Otol

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In terms of pathophysiology, an anatomically narrow airway is a predisposing factor for obstruction of the upper respiratory tract. The correlation between the nasopharyngeal airway and the craniofacial structures is discussed in this context. Thus a mutual interaction between the pharynx and the mandibular position was demonstrated, whereby the transverse dimension of the nasopharynx was significantly larger in patients with prognathism than in patients with retrognathism. The influence of chronic obstruction of the nasal airway on craniofacial development was also discussed. The form-and-function interaction, which ought to explain the causal relationship between nasal obstruction and craniofacial growth, appears to be of a multifactorial rather than a one-dimensional, linear nature. It is not disputed, however, that expanding the maxilla improves not only nasal volume and nasal flow, but also the subjective sensation of patients, although it is not possible to make a prognostic statement about the extent of this improvement because of the differing reactions of individuals. Orthodontic appliances for advancing the mandible can also be successfully used in the treatment of mild obstructive sleep apnea syndrome. This treatment method should be considered particularly for patients who are unwilling to undergo or cannot tolerate CPAP treatment.

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Long face: New proposals for taxonomy, diagnosis, treatment

Cited by 9 publications

References 6 publications

Morphologic determinants in the etiology of class III malocclusions: A review

Morphologic determinants in the etiology of class III malocclusions: A review

Morphologic determinants in the etiology of class III malocclusions: A review

Interaktion von Hals-Nasen-Ohren-Heilkunde und Kieferorthopädie: Die Beziehung zwischen dem nasopharyngealen Luftraum und den kraniofazialen Strukturen

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