“…40,41 In FCHL, the increased CVD risk in probands and first-degree relatives is largely attributed to the increase in apoB 42 and/or lipoprotein (a). 43 Likewise, in dysbetalipoproteinemia, the increased CVD risk is attributed to increased remnant lipoprotein particles. 37 44 However, recent statements by both the Endocrine Society and American Heart Association conclude that although there is growing evidence to support HyperTG is an independent CVD risk, the extent to which it is directly atherogenic remains unclear.…”