Lipid Nephrosis

Schwarz, Herman; Kohn, Jerome L.; Weiner, Samuel

doi:10.1001/archpedi.1943.02010150003001

Cited by 18 publications

(2 citation statements)

References 6 publications

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“…In a series of 208 patients published by Barness et al (1950) 50% of the patients showed complete recovery when seen two to 23 years after the initial illness. Schwartz, Kohn and Weiner (1943) studied 40 patients for periods up to 20 years and report that 45% recovered completely. Farr (1943), studying 36 patients reported a recovery rate of 28%, whereas Greenman, Weigand and Danowski (1955) reported that 22 out of 30 patients (73%) were entirely normal, but the follow-up period extended only for two years.…”

Section: Results Of Therapymentioning

confidence: 99%

The Natural History of Nephrosis

Rm¹

1957

Archives of Disease in Childhood

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Section: Results Of Therapymentioning

confidence: 99%

The Natural History of Nephrosis

Rm¹

1957

Archives of Disease in Childhood

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“…Although hyperlipemia in nephrosis and in the nephrotic stages of glomerulonephritis has been recognized for many years little has been published on this subject recently even in articles on lipid nephrosis (123). peters & Man (124) measured the serum lipids on one hun dred and forty-two occasions in fifty-four patients with glomerulo nephritis.…”

Section: Factors Affecting Blood and Tissue Fatsmentioning

confidence: 99%

Fat Metabolism

Gildea¹,

Man²

1945

Annu. Rev. Biochem.

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This review covers some aspects of fat metabolism which have not been discussed in detail in previous years. It has been difficult to ob tain foreign literature and therefore the review is incomplete. NERVOUS SYSTEM AND FAT METABOLISMRelation of the hypothalamus to fat metabolism.-Since 1940 when the symposium on the hypothalamus was published by the Association for Research in Nervous and Mental Diseases (1), many groups of investigators have made systematic attacks on the problem of hypo thalamic regulation of metabolism including that of fat. Hetherington & Ranson (2, 3, 4) have demonstrated conclusively in rats that care fully placed bilateral lesions in the ventromedial nuclei of the hypothal amus (injury to pituitary was avoided) produced promptly increase of appetite and subsequently extreme obesity. Removal of the pituitary provoked no obesity, and also did not prevent development of obesity following hypothalamic lesions. Even when sufficient time after hypo physectomy, eleven weeks, was allowed to elapse for atrophy of thy roid, adrenals, and gonads to develop, subsequent hypothalamic lesions were still followed by hyperphagia and obesity. These results have been confirmed in the monkey by Brooks, Lambert & Bard (5), in the dog by Heinbecker et at. (6) , and in the cat by Wheatley (7). Hetherington (4) has tried, in an extensive series of experiments, to localize the lesions which consistently result in obesity. He con cluded that cell groups of basal forebrain rostral to the ventromedial hypothalamic nucleus made little if any contribution to regulation of fat metabolism. Destruction of the ventromedial hypothalamic nucleus or its descending fibers in the brain stem was required to produce hyperphagia and obesity.That diffuse injuries to brain may disturb fat metabolism has been suggested by the effects of insufflation of air into ventricles of the dog. Schrade (8) observed that three to four hours after air injection the total blood fats were reduced in amount. Phospholipids, cholesterol, and neutral fats all decreased in concentration. The change in cho-333 Annu. Rev. Biochem. 1945.14:333-356. Downloaded from www.annualreviews.org by Massachusetts Institute of Technology (MIT) on 11/05/14. For personal use only. Quick links to online content Further ANNUAL REVIEWS

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Effect of Measles on the Nephrotic Syndrome

Blumberg¹,

Cassady²

1947

Arch Pediatr Adolesc Med

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Lipid Nephrosis

Cited by 18 publications

References 6 publications

The Natural History of Nephrosis

The Natural History of Nephrosis

Fat Metabolism

Effect of Measles on the Nephrotic Syndrome

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