Links between Monthly Rates of Four Subtypes of Acute Myocardial Infarction and Their Corresponding Cosmophysical Activity Parameters

Abstract

The monthly rates of all subtypes of AMI were significantly correlated with CRA and inversely correlated with SA and GMA, more strongly in female patients. We presume that the environmental factors studied here affect the general patho-physiological components of AMI, and that different subtypes are a consequence of the localization and extent of the process.

Acute Myocardial Infarction (AMI) and Intermediate Coronary Syndrome (ICS)

“…[26] In addition, some of risk factors of Coronary Heart Disease like arterial pressure, blood lipids and coagulation, C-reactive protein (CRP), life threatening cardiac arrhythmia (ventricular fibrillation, tachycardia), atrial fibrillation, cardiac arrhythmia in patients with AMI were studied in relation to accompanying levels of GMA, CRA, SA, Space Proton flux of different energy levels. [1][2][3][4][5][6][7][8][9][26][27][28][29][30][31][32].…”

“…These symptoms can be short, resulting in angina pectoris attacks (AP), and longer and more severe, but remaining without significant myocardial damage named Intermediate Coronary Syndrome (ICS) The drop of blood supply and provoked oxygen and other vital agents disballance for a longer time (mostly more than 20 minutes) often lead to necrobiosis in the area of the damaged (culprit) artery with a specific additional complex of symptoms:(clinical, electrocardiographic, enzymatic) and result in later scar formation, or myocardial rupture, life threatening arrhythmias, aneurysm formation, heart failure that is considered as Acute myocardial Infarction (AMI).The ICS takes place between AP and AMI. Recent studied demonstrated significant links between a number of pathogenetic factors involved in ACS development and environmental physical activity [1][2][3][4][5][6][7][8][9]. The aim of this study was to explore AMI and ICS (code I21-I22 versus I20, ICD 10) by differences in concomitant environmental physical conditions: Geomagnetic, Solar and Cosmic Ray (Neutron) activity.…”

Days of “Zero” level geomagnetic activity accompanied by the high neutron activity and dynamics of some medical events—Antipodes to geomagnetic storms

“…Such results were seen for SCD in a number of studies [15,21,26,27], different forms of cardiac arrhythmia (some forms of them are predictors or components of SCD), also for atrial fibrillation (not only a significant cardiac lesion, but a major risk factor for stroke) [31][32][33][34][35][36]. In a number of following observations also significant links were seen for total number of AMI and its different subtypes (STEMI, NSTEMI, Q-wave and non-Q-wave AMI) [21,35]. Similar findings were published for great groups of cerebral stroke (CVA) [16].…”

“…Death in most cases is a result of fatal cardiac arrhythmia or heart standstill-asystolia. A strong trend of SCD (mostly result of fatal cardiac arrhythmia-ventricular tachycardia, fibrillation) occurred more often on days of high CRA (neutron activity) [25,26,34,35,40].…”

“…It is presumed that high neutron activity, accompanying the lowest GMA level can affect electrical heart instability, a basis for cardiac arrhythmia, and also, is an additional risk factor for unstable (result of disturbed lipid metabolism in the artery wall) atheroma ruptures and fissuring-a basis for the latest stage of the intravascular catastrophic-complete occlusion by thrombus in the region of atheroma plaque disruption and developing infarction in the blocked from oxygen and other life support supply in the zone of "culprit" artery location (heart muscle, brain). One of presumed ways of neutron activity action on the mentioned organ on cellular-molecular levels can be their addiction to H + ions, especially high distributed in lipid concentration places (unstable atheroma and similar locations), conversion neutrons (after joining the H + ion) to protons, that aggressively attacks cell nuclei and other cells parts and can act as one of factors for fatal consequences (SCD, myocardial ruptures in AMI, fatal arrhythmias in ischemic cardiomyopathies and repeated AMI) [25,35,[42][43][44].…”