Abstract:This dissertation is focused on factors that increase risk for nonalcoholic fatty liver disease (NALFD) and the more advanced form nonalcoholic steatohepatitis (NASH). These factors include postprandial lipid handling (chapter II); ceramide (CER) turnover (chapter III), hepatic and mitochondrial CER content (chapter III); glucose turnover (chapter IV), de novo lipogenesis (chapter IV), and hepatic fatty acid oxidation (chapter V). With regard to postprandial lipemia, healthy men consumed an oral isotope (2H11-… Show more
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