Letter: Preleukaemic syndrome and marrow hypoplasia.

Shinton, N. K.; Wells, Donald B.

doi:10.1136/bmj.1.5901.246-c

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2019

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“…Exposure to hypoxia leads to higher production of factor-1a and Fos-like antigen (FOSL) 2, resulting in up-regulation of leptin expression in the EAT, along with increased vascularization, inflammation, and fibrosis [ 16 ]. It has been demonstrated that EAT thickness has a high correlation with sympathetic nervous system hyperactivity and it is a local source of catecholamines in HF patients, which suggests sympathetic nervous system hyperactivity is a potential pathophysiological link between sleep apneas and EAT in HF [ 38 ].…”

Section: Discussionmentioning

confidence: 99%

Left ventricular remodeling and dysfunction in obstructive sleep apnea

Liu

et al. 2019

Herz

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Background. Obstructive sleep apnea syndrome (OSAS) is associated with cardiovascular mortality and morbidity. Several studies have reported that it affects the left ventricle; however, large randomized controlled trials are lacking. The current study aimed to summarize the association between OSAS and left ventricular (LV) structure and function. Methods. Electronic databases (PubMed, Embase, and Cochrane) and references were searched for articles published until March 2018. A systematic review and metaanalysis were performed to assess LV structure and function in OSAS patients based on echocardiography.

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Section: Discussionmentioning

confidence: 99%

Left ventricular remodeling and dysfunction in obstructive sleep apnea

Liu

et al. 2019

Herz

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Letter: Preleukaemic syndrome and marrow hypoplasia.

Cited by 1 publication

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Left ventricular remodeling and dysfunction in obstructive sleep apnea

Left ventricular remodeling and dysfunction in obstructive sleep apnea

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