Abstract:Left-ventricular non-compaction (LVNC) represents an arrest in the normal process of myocardial compaction, resulting in multiple, prominent, persistant trabeculations and deep inter-trabecular recesses communicating with the ventricular cavity. LVNC is a rarely encountered cardiomyopathy and few cases have been reported in pregnancy. In this case report we present a patient who referred to our clinic with symptoms of heart failure during pregnancy and whose echocardiographic examination revealed prominent tra… Show more
“…Increased blood volume and cardiac output may precipitate heart failure symptoms such as dyspnea, dizziness, pedal edema, and orthopnea during pregnancy [12]. Kilic and colleges suggested if the symptoms develop suddenly or become serious further evaluation is needed [5]. In our case, it was not possible to determine this clinical presentation associated with LVNC or peripartum cardiomyopathy.…”
Section: Discussionmentioning
confidence: 76%
“…The patient had attributed her symptoms only mild during pregnancy and increased gradually until first month after delivery. LVNC is diagnosed by two-dimensional and color Doppler echocardiography [5]. Echocardiographic features described include multiple predominant ventricular trabeculations, multiple deep intratrabecular recesses communicating with the ventricular cavity visualized with color Doppler, ventricular dilatation, and reduced systolic and diastolic ventricular function [6].…”
Section: Discussionmentioning
confidence: 99%
“…Echocardiography and Magnetic resonance imaging (MRI) usually use for diagnosis [3]. The presentation may follow heart failure, thrombus embolism, and ventricular arrhythmias [4] and is associated with poor prognosis [5]. LVNC with pregnancy is rare to observe [6].…”
Left-ventricular non-compaction (LVNC) cardiomyopathy is an arrest in the normal process of myocardial compaction and few cases have been reported just after pregnancy. In this case report, we present a patient who had heart failure symptoms at first month of after 38 weeks pregnancy. Echocardiography revealed prominent trabeculation and deep intertrabecular recesses at the left ventricular with an impaired ejection fraction. There is no specific treatment for LVNC just after pregnancy besides the usual management of dilated cardiomyopathy.
“…Increased blood volume and cardiac output may precipitate heart failure symptoms such as dyspnea, dizziness, pedal edema, and orthopnea during pregnancy [12]. Kilic and colleges suggested if the symptoms develop suddenly or become serious further evaluation is needed [5]. In our case, it was not possible to determine this clinical presentation associated with LVNC or peripartum cardiomyopathy.…”
Section: Discussionmentioning
confidence: 76%
“…The patient had attributed her symptoms only mild during pregnancy and increased gradually until first month after delivery. LVNC is diagnosed by two-dimensional and color Doppler echocardiography [5]. Echocardiographic features described include multiple predominant ventricular trabeculations, multiple deep intratrabecular recesses communicating with the ventricular cavity visualized with color Doppler, ventricular dilatation, and reduced systolic and diastolic ventricular function [6].…”
Section: Discussionmentioning
confidence: 99%
“…Echocardiography and Magnetic resonance imaging (MRI) usually use for diagnosis [3]. The presentation may follow heart failure, thrombus embolism, and ventricular arrhythmias [4] and is associated with poor prognosis [5]. LVNC with pregnancy is rare to observe [6].…”
Left-ventricular non-compaction (LVNC) cardiomyopathy is an arrest in the normal process of myocardial compaction and few cases have been reported just after pregnancy. In this case report, we present a patient who had heart failure symptoms at first month of after 38 weeks pregnancy. Echocardiography revealed prominent trabeculation and deep intertrabecular recesses at the left ventricular with an impaired ejection fraction. There is no specific treatment for LVNC just after pregnancy besides the usual management of dilated cardiomyopathy.
“…Its prevalence is unknown, and evidence regarding practice during pregnancy is limited. 200 The clinical picture is highly variable, ranging from asymptomatic patients to patients with refractory HF and severe arrhythmias. There is no specific treatment for non-compaction cardiomyopathy, and therapeutic conduct should be supported by experience with other cardiomyopathies.…”
Note: These statements are for information purposes and should not replace the clinical judgment of a physician, who must ultimately determine the appropriate treatment for each patient.
“…Cuatro madres tenían ventrículo no compacto, pero en ninguna se describió síndrome en espejo (32). Posterior a esta publicación se identificaron otros 3 reportes de VNC en gestantes (33)(34)(35), sin compromiso fetal por esta condición.…”
Objetivo: reportar el caso de una gestante con síndrome en espejo asociada a miocardiopatía no compactada, tanto en la madre como el feto, en los que el tratamiento médico antenatal en la madre llevó a un resultado materno perinatal favorable.
Presentación del caso: se describe el caso de una primigestante de 16 años, con 33 semanas de embarazo, remitida desde una institución de primer nivel de atención a una institución privada de cuarto nivel en la ciudad de Medellín, Colombia, por presentar feto con hidropesía en ultrasonido obstétrico de control. Durante la hospitalización, la paciente presentó signos clínicos y ecocardiográficos de falla cardiaca (disnea, edema e hipoxemia), a la vez que se confirmó el diagnóstico de Hydrops fetalis (síndrome en espejo). Se instauró tratamiento diurético con furosemida en la madre, logrando mejoría del cuadro materno y del edema fetal. En el puerperio mediato hospitalario se confirmaron la presencia de miocardiopatía no compactada en la resonancia magnética nuclear cardiaca, tanto de la madre como del recién nacido. Ambos egresaron en adecuadas condiciones y fueron vinculados al programa de seguimiento cardiovascular: falla cardiaca y de cardiopatía congénitas, respectivamente.
Conclusión: se presenta un caso de síndrome en espejo asociado a miocardiopatía no compactada materna y fetal. Es limitado el número de reportes de síndrome en espejo por anomalías cardiacas (maternas y fetales) y pobre la descripción de los tratamientos realizados que surgen como temas a investigar. Sería importante considerar el diagnóstico de MNC en fetos con hidropesía no asociados a isoinmunización y con disfunción cardiaca, así como su atención por equipos multidisciplinarios.
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