Left sided gallbladder and liver without situs inversus and recurrent sigmoid volvulus after alleged previous total sigmoid resection; a case report and review of literatures

Bayileyegn, Nebiyou Simegnew

doi:10.1016/j.ijscr.2023.107906

Cited by 1 publication

(4 citation statements)

References 13 publications

(17 reference statements)

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“…[24] Total or partial situs inversus is a rare causative factor in SV, in which cases, a long sigmoid mesentery is the causative factor. [68] Multiple-organ anomalies also may be associated with SV. [114] A wandering spleen was reported by Hamdy et al [116] as a cause of SV due to the compression of the ectopic spleen on the rectosigmoid junction.…”

Section: Other Entitiesmentioning

confidence: 99%

“…[32] However, such anatomical variations may be a result, which arises from chronic enlargement of the sigmoid colon. [67] Similarly, Bayileyegn [68] reported a case with left-sided liver. In this patient, free field arising from uncommon hepatic localization may cause the replacement of the sigmoid colon to the right upper abdominal quadrant resulting in SV.…”

Section: Pathological Entitiesmentioning

confidence: 99%

“…[74] Ahmadinejad et al [75] reported a patient with SV arising from an undescended testicle as the first case in the literature, and according to authors, adhesions between spermatic cord and sigmoid mesentery are the causative factor. Bayileyegn [68] reported a case with left-sided gallbladder, whose pathogenesis is unclear.…”

Section: Pathological Entitiesmentioning

confidence: 99%

“…Total or partial situs inversus is a rare causative factor in SV, in which cases, a long sigmoid mesentery is the causative factor. [68] Multiple-organ anomalies also may be associated with SV. [114]…”

Section: Pathological Entitiesmentioning

confidence: 99%

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Sigmoid volvulus and concomitant clinical entities

Atamanalp,

Disci,

Peksoz

2024

Formos J Surg

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Sigmoid volvulus (SV) is common in elders. For this reason, most geriatric comorbidities accompany SV. Additionally, SV may associate with a great number of physiological or pathological entities. The purpose of this review is to evaluate the kind and the incidence of such comorbidities as well as to discuss their pathophysiology. To obtain all SV-related reports in worldwide data, an electronic search of the last 57-years’ literature (from 1967 to date) was made in Web of Science and PubMed databases by using ‘sigmoid volvulus’ heading. Additionally, we evaluated our results including 1,071 patients treated in 57-year period from June 1966 to July 2023. We noted comorbid entities in each case. Tens of physiological or pathological clinical entities may accompany SV. Some entities may be the cause or result of SV, while some others may mimic SV or mimicked by this disease. Moreover, some factors may predispose to SV, while some others may initiate it. Some of these comorbidities have cause and effect relations, while some others are coincidental. However, the relationship between SV and some comorbid entities as well as underlying pathologies are not clear enough.

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Section: Other Entitiesmentioning

confidence: 99%