“…Underlying neurobiological mechanisms have been proposed as moderating this increased risk, as FHP youth often perform differently than FHN controls on tests of neurocognitive function. For example, nonabusing FHP adolescent males performed worse than FHN boys on tests of language functioning and academic achievement (Giancola et al, 1993; Hegedus et al, 1984; Najam et al, 1997; Poon et al, 2000; Sher et al, 2000; Tarter et al, 1984; Viken et al, 1999), organization of new information (Peterson et al, 1992), executive cognitive functioning (Giancola et al, 1996; Harden and Pihl, 1995), perseveration (Giancola et al, 1993), working memory (Corral et al, 1999; Harden and Pihl, 1995; Ozkaragoz et al, 1997), nonverbal memory (Sher et al, 1991), visuospatial skills (Berman and Noble, 1995; Corral et al, 1999; Garland et al, 1993; Ozkaragoz and Noble, 1995; Ozkaragoz et al, 1997; Sher et al, 1991) and attention (Tarter et al, 1989). Multigen-erational transmission (Conrod et al, 1995; LeMarquand et al, 1999; Peterson et al, 1996; Pihl and Bruce, 1995), high familial density (Hill et al, 2000a), early AUD onset in father (Tarter et al, 1989), active paternal alcoholism (Ozkaragoz et al, 1997), and genotypic features (Berman and Noble, 1995) increase the link between FHP and cognitive functioning.…”