“…Cases showed some degree of periportal fibrosis to severe fibrosis [1, 2, 5, 7, 29]. Progression to rapid onset of cirrhosis was evident in the biopsy specimens in cases particularly with autoimmune diseases [24–27, 30, 32–37, 39, 45, 46], and submassive to massive necrosis of liver parenchyma were seen in cases with HAV, paramyxo virus infection, those with positive EBV serology, hypereosinophilia, and post transplant recurrence of PIGCH [1, 3, 24–26, 32–35, 38–41]. …”