2004
DOI: 10.1016/j.psychres.2004.05.025
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Is the biological nature of depressive symptoms in borderline patients without concomitant Axis I pathology idiosyncratic? Sleep EEG comparison with recurrent brief, major depression and control subjects

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Cited by 23 publications
(26 citation statements)
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“…We also found that BPD patients without current comorbid depression and prisoners experienced significantly less SWS than CCs. This may be associated with increased alcohol consumption in patients with BPD (Lester, Rundell, Cowden, & Williams, 1973) or an overlap between BPD and psychotic symptoms (De la Fuente et al, 2004). Our hypotheses remain tentative, however, as these findings were not robust across the majority of BPD groups.…”
Section: Comparisons With Clinical Controls (Ccs)mentioning
confidence: 73%
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“…We also found that BPD patients without current comorbid depression and prisoners experienced significantly less SWS than CCs. This may be associated with increased alcohol consumption in patients with BPD (Lester, Rundell, Cowden, & Williams, 1973) or an overlap between BPD and psychotic symptoms (De la Fuente et al, 2004). Our hypotheses remain tentative, however, as these findings were not robust across the majority of BPD groups.…”
Section: Comparisons With Clinical Controls (Ccs)mentioning
confidence: 73%
“…Reasons for non-inclusion in the meta-analysis included: the reporting of overlapping data (De la Fuente et al, 2004); insufficient data to convert into a common effect size (Bromundt et al, 2013); and the uniqueness of the sleep factor (Dagan, Stein, Steinbock, Yovel, & Hallis, 1998), i.e., it wasn't assessed in a minimum of two studies.…”
Section: Search Resultsmentioning
confidence: 99%
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“…Initially, it was debated whether observations of shortened REM latency in BPD patients should be taken as evidence that the disorder was best classified as a mood disorder rather than a personality disorder [18][19][20][21]54]. Shortened REM latency is often found in MDD [22], and it has been argued that this finding suggests a common biological etiology for BPD and mood disorders (e.g., [18,23,24]). However, with the increased research interest in BPD over the years, and discrepant findings in the literature on BPD and sleep, this research has become focused on understanding what it is about BPD that might cause sleep disturbance, rather than what this relationship can tell us about how the disorder should be classified.…”
Section: Polysomnographic Studiesmentioning
confidence: 99%
“…De la Fuente et al, 2004;Nigg, Lohr, Westen, Gold, & Silk, 1992; Q6 Riso, Klein, Anderson & Ouimette, 2000; Soloff et al, 2000; Sprock, Rader, Kendall, & Yoder, 2000; White, Flanagan, Martin, & Silvermann, 2011.…”
mentioning
confidence: 99%