“…However, an investigation of multimodal emotion processing in the disorder is justified in light of evidence suggesting that it is subserved by neural structures that are implicated in the pathophysiology of BD, including the temporal lobe, amygdala, anterior cingulate, and prefrontal cortex (de Gelder, Böcker, Tuomainen, Hensen, & Vroomen, 1999;Dolan, Morris, & de Gelder, 2001;Laurienti et al, 2003;Phillips, Drevets, Rauch, & Lane, 2003;Phillips, Ladouceur, & Drevets, 2008;Pourtois, de Gelder, Bol, & Crommelinck, 2005). This, coupled with the large literature indicating that emotion perception from facial, and to a lesser extent prosody is impaired in the disorder, and evidence suggesting that multimodal integration itself is deficient in the genetically and phenotypically related disorder schizophrenia (Craddock, O'Donovan, & Owen, 2006, 2005de Gelder et al, 2005;Kohler, Walker, Martin, Healey, & Moberg, 2010;Rossell et al, 2013;Van Rheenen & Rossell, 2013a, 2013b, 2014, certainly provides a foundation from which it is reasonable to assume that abnormalities in crossmodal influences between different sensory modalities may be present in BD.…”