Intravascular monitoring in acute Myocardial Infarction

Hunt, D; Pombo^Ramos, Joaquin F.; Potanin, Constantine; Russell, Richard O.; Rackley, Charles E.

doi:10.1016/0002-9149(70)90880-5

Cited by 11 publications

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“…However, measurements which have been made in patients with left heart failure (Yu, 1969) and in a few patients with myocardial infarction (Kirby et al, 1968;Bradley et al, 1970) have shown PVR to be either normal or only slightly elevated. There is also accumulating evidence to show that LAP and left ventricular end-diastolic pressure is raised in a large number of patients with myocardial infarction (Kirby et al, 1968;Nixon, 1968;Begg et al, 1969;Hodges et al, 1969;Russell et al, 1969;Bradley et al, 1970;Hunt, Pombo Ramos, Potanin, Russell & Rackley, 1970). As a result of these studies it would seem reasonable to assume that the raised PAP in our patients is essentially reflecting raised LAP resulting from left ventricular failure.…”

Section: Discussionmentioning

confidence: 58%

Relationship between Haemodynamic and Respiratory Function in Patients with Myocardial Infarction and Left Ventricular Failure

1972

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1.Forty-five patients with myocardial infarction and clinical signs of left ventricular failure have been studied with measurements of pulmonary and brachial artery pressure, cardiac output, arterial blood gas pressures and expired air collections.2. Pulmonary artery pressure was elevated and stroke index was reduced in the majority of patients (89% and 85% respectively). There was a close inverse correlation between these two measurements, both of which appear to be direct consequences of impaired left ventricular function.3. Arterial hypoxaemia was common and only transiently relieved by hyperventilation. Dead space/tidal volume ratio and venous admixture were both increased, compatible with considerable ventilation/perfusion mismatching in the lung. Both measurements correlated directly with pulmonary artery pressure.Pulmonary oedema is a common complication of myocardial infarction. Methods of assessment are mainly indirect and the severity varies from that of early interstitial oedema, which is difficult to detect by clinical and radiological methods, to that of florid and rapidly fatal alveolar oedema. Arterial hypoxaemia has been found in the majority of patients studied and has been associated with evidence of impaired gas exchange in the lungs (Mackenzie, Taylor,

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Section: Discussionmentioning

confidence: 58%

Relationship between Haemodynamic and Respiratory Function in Patients with Myocardial Infarction and Left Ventricular Failure

1972

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“…of left ventricular end-diastolic pressure in acute myocardial infarction (Rapaport and Scheinman, 1969;Hunt, Pombo, Potanin, Russell, and Rackley, 1970). However, this, too, has been reported frequently to underestimate left-ventricular end-diastolic pressure, especially when the latter is significantly elevated in acute myocardial infarction (Lal, Loeb, Sinno, Rahimtoola, Chuquimia, Rosen, and Gunnar, 1971).…”

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confidence: 99%

Right and left heart pressures in acute myocardial infarction

Weisse

Narang

Haider

et al. 1973

Cardiovascular Research

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AUTHORS' SYNOPSIS In dogs the effects of acute myocardial infarction (AMI) on right and left heart pressure relationships were studied. Right atrial pressure was a poor predictor of left heart pressures. Pulmonary artery end-diastolic, left atrial, and left ventricular end-diastolic pressures tended to equalize in normals (except with tachycardia) and after AMI. In a previously scarred left ventricle, however, after superimposed AM1 and marked elevations in left ventricular end-diastolic pressure, prominent atrial impact waves resulted in this being appreciably higher than the other two pressure measurements.Although pressure relationships between the right and left sides of the heart have long been of interest to cardiovascular physiologists, increasing utilization of right heart pressures in monitoring patients with acute myocardial infarction for indications of left-sided events underscores the need for a better appreciation of the uses and limitations of such measurements.While central venous pressure tends to rise with elevations of left atrial and ventricular enddiastolic pressures, use of the central venous pressure as an indicator of the level of left heart pressures in acute myocardial infarction has been unrewarding (Forrester, Diamond, McHugh, and Swan, 1971;Hamosh and Cohn, 1971). Since, in the absence of mitral stenosis and pulmonary vascular obstruction, the pulmonary artery end-diastolic pressure has been shown to reflect the left ventricular end-diastolic pressure in stable patients studied at catheterization (Kaltman, Herbert, Conroy, and Kossman, 1966), this has been recommended as a better indicator

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“…Ή άνάπτυξις και ευρεία διάδοσις τών μονάδων εντατικής παρακολου θήσεως (intensive care units), συνετέλεσεν εις τήν σημαντικήν ελάττωσιν τών θανάτων εκ διαταραχών τοΰ ρυθμού, κατά τήν όξεϊαν φάσιν του εμφράγματος (14,30,34,42,44,47). Ai μονάδες αυταί παρέχουν τήν δυνατότητα εγκαίρου άνακαλώψεως επικινδύνων αρρυθμιών, αϊτινες εν συνεχεία αντιμετωπίζονται πολλάκις επιτυχώς δια φαρμάκων ή ηλεκτρικού shock.…”

Section: εισαγωγηunclassified

Αιμοδυναμικη Μελετη Του Οξεος Εμφραγματος Του Μυοκαρδιου

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Intravascular monitoring in acute Myocardial Infarction

Cited by 11 publications

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Relationship between Haemodynamic and Respiratory Function in Patients with Myocardial Infarction and Left Ventricular Failure

Relationship between Haemodynamic and Respiratory Function in Patients with Myocardial Infarction and Left Ventricular Failure

Right and left heart pressures in acute myocardial infarction

Αιμοδυναμικη Μελετη Του Οξεος Εμφραγματος Του Μυοκαρδιου

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