We review insights into slipped capital femoral epiphysis (SCFE) gained during the last decade and updates to current management practices. Anatomic and clinical studies recently demonstrate that the epiphysis rotates around an epiphyseal tubercle during displacement. Clinical endocrinopathies contribute to the pathogenesis of SCFE, and recently the effects of subclinical endocrine derangements have been demonstrated to play a role in SCFE. Patients with positive age-weight or age-height testing are recommended to undergo further endocrine workup due to the high likelihood of atypical SCFE in these patients. In situ pinning with two screws is mainly reserved for unstable or severe slips, while one-screw fixation remains the standard for mild-moderate slips. Contralateral prophylactic pinning is typically considered in those patients with an atypical slip, relative skeletal immaturity, or aberrant radiographic parameters such as posterior epiphyseal tilt or sloping angle. Novel intraoperative epiphyseal perfusion monitoring has provided insight into reducing complications such as avascular necrosis and has shown the benefit of intracapsular hematoma decompression for unstable SCFE. Open surgical management via the modified Dunn procedure should be cautiously considered, as high rates of osteonecrosis have been reported due to the vulnerable blood supply of the proximal femoral head.
Key Concepts• The epiphyseal tubercle acts as a prominent stabilizer of the proximal femoral epiphysis and pivot point for a posterior rotation and displacement of the epiphysis during SCFE.• Hyperinsulinism and increased leptin concentrations lead to biochemical alterations of the proximal femoral physis that contribute to physeal failure.• Further endocrine and metabolic workup is needed for patients with a positive age-weight or age-height test due to the high likelihood of atypical SCFE.