Intracranial Hypertension Resulting From a Cerebrovascular Malformation

Weisberg, Leon A.; Pierce, Jordan; Jabbari, Bahman

doi:10.1097/00007611-197705000-00040

Cited by 28 publications

(4 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…This may increase cerebral blood volume and produce raised intracanial pressure. Second, high venous pressure due to the fistula may result in a reduction of CSF absorption across the arachnoid villi with subsequent high CSF pressure (Weisberg et al 1977, Barrow 1988, Chimowitz et al 1990.…”

Section: Discussionmentioning

confidence: 99%

Benign intracranial hypertension associated with arteriovenous malformation

Vorstman¹,

Niemann²,

Molyneux³

et al. 2002

Develop Med Child Neuro

View full text Add to dashboard Cite

This is a report of a 9‐year‐old female with a clinical presentation of benign intracranial hypertension (BIH) who was found to have an unruptured pial arteriovenous malformation (AVM) with a significant fistula. The AVM was completely embolized using a recently developed liquid embolic system, Onyx, after which gradual clinical improvement followed. A few cases of BIH associated with AVM have been described in adults and adolescents. Possible causal relation is discussed.

show abstract

Section: Discussionmentioning

confidence: 99%

Benign intracranial hypertension associated with arteriovenous malformation

Vorstman¹,

Niemann²,

Molyneux³

et al. 2002

Develop Med Child Neuro

View full text Add to dashboard Cite

show abstract

“…Presenting symptoms included headache (6 patients); transient language, motor, or sensory symptoms (4); visual obscurations (3); focal seizures (2); and progressive bilateral visual loss (1). Signs included papilledema (6 patients), ipsilateral carotid or ocular bruits (3), and abnormal visual fields (2 had bilateral inferior nasal field loss and 1 had left nasal and right temporal scotomas). Only 1 patient had a lumbar puncture, which yielded an opening pressure of 28 cm H20.…”

Section: Methodsmentioning

confidence: 99%

Intracranial hypertension associated with unruptured cerebral arteriovenous malformations

Chimowitz

Little

Awad

et al. 1990

Annals of Neurology

View full text Add to dashboard Cite

Only 6 patients with intracranial hypertension associated with unruptured cerebral arteriovenous malformations have been reported. We report 6 additional patients seen at the Cleveland Clinic during the past 10 years. The average age was 28 years (range, 19-44 years); 4 were women. Symptoms and signs included papilledema (6 patients), headache (6), transient nonepileptic focal symptoms (4), visual obscurations (3), ipsilateral carotid or ocular bruits (3), abnormal visual fields (3), focal seizures (2), and progressive visual loss (1). Enhanced computed tomography (CT) or magnetic resonance imaging (MRI) demonstrated the malformations in all 6 patients. The malformations were large, supplied by the branches of the middle and anterior cerebral arteries, with the posterior cerebral artery contributing in 3 patients, and all drained into the superior sagittal sinus. Associated venous obstruction was seen in 2 patients. Four patients underwent excision of the arteriovenous malformation, with resolution of papilledema in all 4. Measurements of cortical arterial and venous pressures during surgery in 3 patients showed decreased feeding artery pressures and elevated draining vein pressures, which normalized after removal of the malformation. Treatment in the 2 remaining patients consisted of medical therapy (acetazolamide, furosemide, steroids) alone in 1 patient, and in conjunction with proton beam radiation in the other. Papilledema resolved in the former patient, but the patient receiving proton beam radiation still had papilledema 2 years later. Intracranial hypertension associated with unruptured cerebral arteriovenous malformations occurs in young patients with high flow malformations that drain into the superior sagittal sinus, and is likely the result of increased cortical venous and superior sagittal sinus pressure. Excision of the malformation effectively reduces the intracranial pressure.

show abstract

“…4 The recognition of arteriovenous malformations as a cause of pseudotumour cerebri would have been expected to increase with the development of magnetic resonance scanning. However, there are only four cases of pseudotumour cerebri in association with arteriovenous malformations in the literature in whom other causes of pseudotumour cerebri including venous sinus occlusion, have been excluded.49 11 We present two further patients who have pseudotumour cerebri secondary to an arteriovenous malformation (AVM) and review the possible mechanism of raised intracranial pressure.…”

Section: Introductionmentioning

confidence: 99%