2004
DOI: 10.1002/bjs.4703
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Intra-abdominal hypertension and the abdominal compartment syndrome

A F K Moore,
R Hargest,
M Martin
et al.

Abstract: The optimal time for intervention is not known, but outcome is often poor, even after decompression. Most of the available information relates to victims of trauma rather than general surgical patients.

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Cited by 115 publications
(101 citation statements)
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“…Thus, cirrhosis induces the development of hyperkinetic circulation, likely to be characterized by tachycardia and increased myocardial ejection fraction [8]. Additionally, ascites, which commonly coexists with cirrhosis, increases intra-abdominal pressure, leading to hemodynamic abnormalities of the myocardium and impairing renal function; moreover, it affects intra-thoracic pressure and can alter the anatomical position of the heart, and therefore its electrical axis [9,10].…”
mentioning
confidence: 99%
“…Thus, cirrhosis induces the development of hyperkinetic circulation, likely to be characterized by tachycardia and increased myocardial ejection fraction [8]. Additionally, ascites, which commonly coexists with cirrhosis, increases intra-abdominal pressure, leading to hemodynamic abnormalities of the myocardium and impairing renal function; moreover, it affects intra-thoracic pressure and can alter the anatomical position of the heart, and therefore its electrical axis [9,10].…”
mentioning
confidence: 99%
“…5 The management of the open abdomen is extremely challenging; early problems include infection and ongoing fluid losses, while fistulae and ventral hernias can lead to long-term complications. 6 There is, therefore, an understandable reluctance amongst surgeons to perform this procedure. The majority of intensivists would recommend surgical decompression when the IAP exceeds 25mmHg and there is organ dysfunction (64.4%, n=67).…”
Section: Discussionmentioning
confidence: 99%
“…IAP is transmitted to the thoracic cavity directly or indirectly thought deviation of the diaphragm. The upward movement of the diaphragm causes reduction of chest wall and lung compliance, increasing intrapleural pressure, decrease of ventilation and increase of pulmonary vascular resistance [10][11][12][13]. The main respiratory problem in IAH patients is the development of compression atelectasis of the lung parenchyma, mainly in the caudal parts near diaphragm, caused by its elevation [14].…”
Section: Introductionmentioning
confidence: 99%
“…The main respiratory problem in IAH patients is the development of compression atelectasis of the lung parenchyma, mainly in the caudal parts near diaphragm, caused by its elevation [14]. As a result, all static and dynamic lung volumes are decreased [12,[15][16][17]. Adequate ventilation may only be achieved byincreased airway pressure.…”
Section: Introductionmentioning
confidence: 99%