Interim report on DMF‐reduction 16 years after the introduction of a preventive program

Marthaler, T

doi:10.1111/j.1600-0528.1981.tb00331.x

Cited by 18 publications

(10 citation statements)

References 7 publications

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“…The slow but continuous progression of approximal caries and the relatively high prevalence of enamel caries at 21 suggest that for approximal surfaces in a long-term perspective, the fluoride supplements simply retard the progression of caries. This is in accordance with Marthaler [1981], who in a report on the effect of water fluoridation observed that while the prevalence of new approximal enamel lesions in 12-to 14-yearolds did not show any substantial decrease, the number of dentin lesions was markedly reduced. Also Groeneveld [1985], comparing the number of approximal enamel lesions in a fluoridated and a non-fluoridated area, in children from 9 to 18, found only a small retardation in the initiation of approximal lesions, while caries progression beyond the stage of enamel caries was markedly reduced in the fluoridated area.…”

Section: Discussionsupporting

confidence: 78%

Caries Development from 11 to 22 Years of Age: A Prospective Radiographic StudyPrevalence and Distribution

Mejàre

Källestål²,

Stenlund³

et al. 1997

Caries Res

142

103

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The aim was to assess the distribution and pattern of caries development longitudinally in teenagers and adolescents whose treatment had been based on remineralizing rather than restorative strategies. A baseline cohort of 536 children was studied, using bite-wing radiographs, from 11 to 22 years of age. The results showed a slow but continuous increase in both enamel and dentin caries of approximal surfaces. At 21, 29% of all posterior approximal surfaces had enamel caries according to the radiographic diagnoses, 14% had dentin caries and another 5% were restored. At the age of 20–21, the skewed distribution of DMFS_appr apparent at 12 and 15 had given way to a more uniform picture and the percentage of individuals with no decayed approximal surfaces (DMFS_appr = 0) decreased from 71 at 12–13 to 28 at 20–21 years of age. The proportion of DFS_occl in relation to all DFS decreased from 83% at 12 to 52% at 21. The occlusal, mesial and distal surfaces of the first molar accounted for 60% of all restored surfaces at 21.

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Section: Discussionsupporting

confidence: 78%

Caries Development from 11 to 22 Years of Age: A Prospective Radiographic StudyPrevalence and Distribution

Mejàre

Källestål²,

Stenlund³

et al. 1997

Caries Res

142

103

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“…Other studies have also observed the limited effect of fluoride exposure to reduce the occurrence of enamel lesions. 29,30 In this study there was also a potential for misclassification of fluoride exposure because exposure was based on the child's reported place of residence rather than a direct measure of exposure. However, the misclassification would have been non-differential misclassification, the effect of which would have been to lead the estimate towards the null effect.…”

Section: Discussionmentioning

confidence: 97%

Incidence and progression of approximal carious lesions among school children in Western Australia

Arrow

2007

Australian Dental Journal

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Background: An understanding of carious lesion behaviour is important in timing and frequency of radiographic examinations. The aim of this study was to examine the occurrence and behaviour of approximal enamel lesions in permanent teeth among children in Western Australia and test the effect of easily measured factors on the rate of lesion progression. Methods: Bitewing radiographs of children enrolled with the School Dental Service in Western Australia were scored for the occurrence and progression of enamel caries using established criteria. Time to lesion occurrence was estimated by the use of the Kaplan-Meier analysis, bivariate tests of survival functions were undertaken using the log rank test and the influence of the measured factors were tested using the multivariate Cox proportional hazard model. Results:The median time to occurrence of enamel caries was 6.1 months; median time to dentine caries was 77.7 months. Multivariate analysis shows gender (hazard ratio, male 0.65), baseline permanent tooth caries experience (hazard ratio 1.15) and time at risk since eruption (hazard ratio 0.88) were important factors in the initiation of enamel caries while time since eruption (hazard ratio 0.87), clinician's rating of risk (hazard ratio, moderate risk 2.78, high risk 5.00) and residence in fluoridated area (hazard ratio, fluoridate 0.44) were all statistically significant for enamel caries progression. Conclusions: Time to occurrence of enamel lesions in approximal surfaces is relatively rapid while progression into dentine is relatively slow. Clinicians were able to assign the risk of lesion progression in a child. Residence in a fluoridated area has a marked retarding effect on enamel lesion progression but not on initiation of enamel lesions. These factors may be useful in developing a schedule for timing and frequency of radiographic examinations among children in Western Australia.Key words: Bitewing radiographs, caries incidence, caries progression, survival analysis.Abbreviations and acronyms: DMFT = decayed, missing and filled permanent teeth; SDS = School Dental Service.

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“…Interestingly, Manji et al showed that the model predicted the apparently enigmatic findings in the Netherlands fluoridation studies [Backer Dirks et al, 1961;Marthaler, 1981;Groeneveld, 1985] where the effect of water fluoridation was most pronounced when caries was diagnosed at cavity level, whereas when early enamel demineralized lesions are included in the diagnosis, there would be no significant differences.…”

Section: The Biofilm and The Response Of The Dental Hard Tissuesmentioning

confidence: 99%

Caries and Periodontitis: Contesting the Conventional Wisdom on Their Aetiology

Manji¹,

Dahlén

Fejerskov

2018

Caries Res

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We review the literature on the oral microbiome and the role of the microbiota in the development of dental caries and periodontitis. While most research has been focused on identifying one or more specific determinants of these diseases, the results have provided limited predictive value and have not been able to explain the variation in the distribution of these diseases observed in epidemiological or clinical studies. Drawing on existing knowledge about the nature of the oral microbiota, we suggest that a stochastic model based on the Weiner process provides simple and parsimonious explanations for the pathogenesis of both caries and periodontitis, making few assumptions, and providing explanations for phenomena that have hitherto proved difficult, or have required complex arguments, to explain. These diseases occur as the result of the dental hard tissues and periodontal tissues integrating the random “noise” caused by normal metabolic activities of commensal microorganisms in the dental biofilm. The processes that result in the progression and regression of caries and periodontitis may be considered as “natural,” rather than pathological, even if, when left unchecked over long periods of time, they can result in the development of pathologies. The likelihood of progression or regression can be influenced by other determinants, but these processes will nevertheless occur in the absence of such influences. The distributional characteristics of the model approximate the findings of epidemiological studies indicating that, for both caries and periodontitis, there will be few sites affected in the early period after the eruption of the permanent dentition, but in those older there is an almost linear relationship with increasing age; furthermore, the longer a site survives without being affected, the less likely that it will be affected. We discuss the clinical and public health importance of these findings.

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Interim report on DMF‐reduction 16 years after the introduction of a preventive program

Cited by 18 publications

References 7 publications

Caries Development from 11 to 22 Years of Age: A Prospective Radiographic StudyPrevalence and Distribution

Caries Development from 11 to 22 Years of Age: A Prospective Radiographic StudyPrevalence and Distribution

Incidence and progression of approximal carious lesions among school children in Western Australia

Caries and Periodontitis: Contesting the Conventional Wisdom on Their Aetiology

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